Is poikiloderma a graft-versus-host-like reaction?

Person, John R.; Bishop, Gregory F.

doi:10.1097/00000372-198402000-00015

Cited by 8 publications

(2 citation statements)

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“…Various degrees of epidermal atrophy, vacuolar alteration ofthe basal layer, lymphohistiocytic infiltrate in the papillary dermis and vascular ectasia have been described (11). The histologic features in our patient were consistent with poikiloderma (12).…”

Section: Discussionsupporting

confidence: 75%

Poikiloderma Congenitale: Case Report and Review of the Literature

Collins

Barnes

McCabe

1991

Pediatric Dermatology

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Section: Discussionsupporting

confidence: 75%

Poikiloderma Congenitale: Case Report and Review of the Literature

Collins

Barnes

McCabe

1991

Pediatric Dermatology

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“…[135] Some poikilodermas may be related, in some ways to a graft versus host reaction. [136] Dermatomyositis Skin lesions have shown a spectrum of histopathological changes, varying from sparse perivascular lymphocytic infiltrate, edema, mucinous changes in the upper dermis, to full-fledged lichenoid reaction pattern with prominent basal vacuolar changes. Occasional civatte bodies and/or neutorphils can be an accompaniment.…”

Section: Lichenoid Eruptionsmentioning

confidence: 99%

Lichenoid tissue reaction/interface dermatitis: Recognition, classification, etiology, and clinicopathological overtones

Sehgal¹,

Srivastava²,

Sharma³

et al. 2011

Indian J Dermatol Venereol Leprol

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Lichenoid tissue reaction or interface dermatitis embrace several clinical conditions, the prototype of which is lichen planus and its variants, drug induced lichenoid dermatitis, special forms of lichenoid dermatitis, lichenoid dermatitis in lupus erythematosus, and miscellaneous disorders showing lichenoid dermatitis, the salient clinical and histological features of which are described to facilitate their diagnosis. Background of lichenoid reaction pattern has been briefly outlined to enlighten those interested in this entity.

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Poikilodermatous Mycosis Fungoides and Atrophic Large-Plaque Parapsoriasis Exhibit Similar Abnormalities of T-Cell Antigen Expression

Lindae

Abel²,

Hoppe³

et al. 1988

Arch Dermatol

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We studied the immunohistologic findings of skin biopsy specimens from 21 patients with poikiloderma (14 with mycosis fungoides [MF] and seven with atrophic large-plaque parapsoriasis [ALPP]). Both types of poikiloderma were similar with regard to T-cell antigen expression. In each case, most T cells expressed the CD4+ (helper/inducer) phenotype and lacked Leu-8 antigen. T cells were also deficient in Leu-9 antigen in most cases (MF, 11/14 [79%]; ALPP, 4/7 [57%]). These T-cell antigen deficiencies are similar to those described previously in various types of MF and indicate that such deficiencies are common in minimally infiltrated, patch-stage MF lesions. Because combined Leu-8/Leu-9 antigen deficiencies are uncommon in inflammatory skin diseases, our findings are consistent with the view that ALPP is an early form of MF, as had been suggested previously by results of clinicopathologic studies.

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Is poikiloderma a graft-versus-host-like reaction?

Cited by 8 publications

References 0 publications

Poikiloderma Congenitale: Case Report and Review of the Literature

Poikiloderma Congenitale: Case Report and Review of the Literature

Lichenoid tissue reaction/interface dermatitis: Recognition, classification, etiology, and clinicopathological overtones

Poikilodermatous Mycosis Fungoides and Atrophic Large-Plaque Parapsoriasis Exhibit Similar Abnormalities of T-Cell Antigen Expression

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