Lung fibrosis and exposure to wood dusts: Two cases report and review of the literature

Riccò, Matteo

doi:10.13075/mp.5893.00140

Cited by 8 publications

(3 citation statements)

References 39 publications

(67 reference statements)

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“…A Swedish multicenter case-control study on occupational exposure revealed a higher IPF risk in males with birch dust and hardwood dust exposure and no increased risk Epidemiology of Idiopathic Pulmonary Fibrosis DOI: http://dx.doi.org/10.5772/intechopen.98482 with metal exposure [49]. A similar increased association was observed in an Italian case series and an Egyptian multicenter case-control study [40,50]. Air pollutants present in the environment may have an important role apart from smoking in IPF incidence.…”

Section: Environmental Factorsmentioning

confidence: 52%

Epidemiology of Idiopathic Pulmonary Fibrosis

Patil¹

2022

Idiopathic Pulmonary Fibrosis

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Idiopathic pulmonary fibrosis (IPF) is a type of interstitial lung disease (ILD) classified under idiopathic fibrotic disorders of the lung. It is the most common type of ILD presenting clinically in the seventh decade of life, almost always at the later stage of illness, attributed to its earlier nonspecific presentation. The term IPF is used when no specific cause for pulmonary fibrosis is identified. Initially described in 1944, recent advances in lung biopsy and pathology have described the disease in detail. This led to further classification of ILD. Also, there have been multiple recent studies indicative of an increased incidence. However, accurate epidemiological data for IPF is minimal, with some being contradictory. Inconsistency in the case definition criteria and methodology has resulted in epidemiological inaccuracy when used to detect patients in the study population. To avoid inaccuracy American Thoracic Society collaborated with the European, Japanese, and Latin American Thoracic Society to arrive at a consensus resulting in 2010 IPF evidence-based guidelines. Notable epidemiological differences are observed in the European, American, and Asian countries. Some countries have set up national registries to collect essential patient data for future studies and comparison with other countries. In this topic, we try to glean over the epidemiology of IPF.

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Section: Environmental Factorsmentioning

confidence: 52%

Epidemiology of Idiopathic Pulmonary Fibrosis

Patil¹

2022

Idiopathic Pulmonary Fibrosis

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“…Additional data supporting the idea that wood dust exposure may play a role in the pathogenesis of IPF has been presented in the form of a case-series from Italy [58] and, as discussed previously, the work from Egypt by Awadalla et al [42], which found that men with a history of carpentry and wood-working with occupational exposure to wood dust and wood preservative had a significantly higher risk of IPF. These studies suggest that occupational exposures to wood dusts may contribute to the development of IPF.…”

Section: Wood Dustmentioning

confidence: 61%

The Role of Occupational and Environmental Exposures in the Pathogenesis of Idiopathic Pulmonary Fibrosis: A Narrative Literature Review

Trethewey

Walters

2018

Medicina

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Idiopathic pulmonary fibrosis (IPF) is a chronic interstitial lung disease characterised by a progressive and irreversible decline in lung function, which is associated with poor long-term survival. The pathogenesis of IPF is incompletely understood. An accumulating body of evidence, obtained over the past three decades, suggests that occupational and environmental exposures may play a role in the development of IPF. This narrative literature review aims to summarise current understanding and the areas of ongoing research into the role of occupational and environmental exposures in the pathogenesis of IPF.

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“…В то же время она имеет защитное значение для других систем и всего организма в це-лом. Слизистая оболочка полости носа не только за-держивает частицы пыли, но и благодаря бактери-цидным свойствам слизи, вырабатываемой бокало-видными клетками, способствует частичному обез-вреживанию бактерий [7][8][9]. Значительное количе-ство нервных окончаний в слизистой оболочке по-лости носа обеспечивает рефлекторную защиту ор-ганизма от многих внешних воздействий, в том числе и от ПА.…”

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Upper respiratory tract diseases caused by exposure to industrial aerosols

Панкова¹,

Федина²,

Nakatis³

et al. 2016

Ross. rinol.

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Актуальность проблемы клинико-функцио-нальных нарушений верхних дыхательных путей (ВДП) обусловлена значительным числом лиц, кон-тактирующих с промышленными аэрозолями (ПА) в различных отраслях экономики. Выделение ПА характерно для таких производственных процессов, как добыча, разрушение, дробление, измельчение, пересыпка веществ, сварка, напыление, наплав и др. Наиболее высокие концентрации пылевого воз-действия наблюдаются в угольной, деревообрабаты-вающей, металлургической, текстильной, хлопко-обрабатывающей, машиностроительной, сельско-хозяйственной отраслях экономики.Инспираторный путь поступления и физико-химические свойства ПА (агрегатное состояние в виде аэрозолей и увеличение летучести при нагреве) приводят к высокой распространенности измене- Цель -систематизировать клинико-патофизиологические изменения слизистой оболочки верхних дыхательных путей (ВДП) у работников, подвергающихся воздействию различных промышленных аэрозолей (ПА). Основные положения. Клиническая картина дистрофических изменений слизистой оболочки ВДП практически не имеет каких-либо специфических черт и развивается по типу катарального, субатрофического или гипертрофического ринита, фарингита, ларингита. Особенностями формирования дистрофического процесса являются нисходящий характер изменений и тотальное поражение всех отделов ВДП (ринофаринголарингит), прогрессирование процесса по мере увеличения стажа работы в условиях воздействия ПА. Ингаляционное поступление ПА в организм создает возможности для воздействия сенсибилизирующего химического вещества на весь респираторный тракт. Заключение. Наличие профессиональных заболеваний ВДП, вызванных воздействием ПА, за исключением аллергической патологии не снижает профессиональной трудоспособности работника, однако требует безотлагательной реабилитации нарушенных функций и полноценного целенаправленного лечения. Objective. To systematize clinical and pathophysiological changes in the upper respiratory tract (URT) mucosa in workers exposed to various industrial aerosols (IAs). The clinical picture of degenerative changes in the URT mucosa has virtually no any specific features and follows the pattern of catarrhal, subatrophic or hypertrophic rhinitis, pharyngitis, and laryngitis. The features of development of a dystrophic process are the descending pattern of changes and the total involvement of all sections of the URT (rhinopharyngolaryngitis), and process progression as the length of practical experience upon exposure to IAs increases. The inhalation of IAs creates opportunities for exposure of the entire respiratory tract to a sensitizing chemical. Occupational URT diseases caused by exposure to IAs, with the exception of allergic pathology, do not reduce occupational disability of a worker; however, requires immediate rehabilitation of impaired functions and adequate targeted treatment.Keywords: industrial aerosols; occupational diseases of the upper respiratory tract; dystrophic, allergic changes in the mucosa of the nose, pharynx, and larynx. Сведения об авторах:Панкова Вера Борисовна...

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Lung fibrosis and exposure to wood dusts: Two cases report and review of the literature

Cited by 8 publications

References 39 publications

Epidemiology of Idiopathic Pulmonary Fibrosis

Epidemiology of Idiopathic Pulmonary Fibrosis

The Role of Occupational and Environmental Exposures in the Pathogenesis of Idiopathic Pulmonary Fibrosis: A Narrative Literature Review

Upper respiratory tract diseases caused by exposure to industrial aerosols

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