Marijuana Induced Acute Coronary Syndrome

Velasquez, Skarlet W. Patino; Calderón, Rafael; Rabelo, Nikita; Patino, Jhonny

doi:10.1016/s0735-1097(20)33836-5

Cited by 1 publication

(2 citation statements)

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“…Marijuana‐induced increase in carboxyhaemoglobin level in blood and subsequent reduction of normal haemoglobin saturation lead to an increased demand for blood supply in the myocardium as a consequence of reduced oxygen supplementation. These changes can lead to coronary vasoconstriction and, in turn, to temporary myocardial ischaemia and angina pectoris symptoms, as well as ischaemic changes in ECG in patients with correct coronary arteries 2,3,5 . These people usually have not had a history of similar ailments in the past.…”

Section: Discussionmentioning

confidence: 99%

“…These changes can lead to coronary vasoconstriction and, in turn, to temporary myocardial ischaemia and angina pectoris symptoms, as well as ischaemic changes in ECG in patients with correct coronary arteries. 2,3,5 These people usually have not had a history of similar ailments in the past. According to the work by Desai et al, the risk of myocardial infarction (MI) increases by 8% in patients who smoke marijuana recreationally.…”

Section: Discussionmentioning

confidence: 99%

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Myocardial ischaemia associated with tetrahydrocannabinol intake in a teenage boy: A case report

Stasiak

Kędziora

2020

J Paediatrics Child Health

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The presented case concerns a 15-year-old-boy who was admitted to the emergency department because of chest pain. According to his medical history, the first episode of an oppressive pain occurred 2 days prior to the admission to hospital and resolved spontaneously after a few hours. On the next day, the boy worked physically and reported another episode of chest pain radiating to the left upper limb, accompanied by shortness of breath and nausea. Due to persisting ailments, the patient was transported to the hospital by an ambulance. In the emergency department, a 12-lead electrocardiogram (ECG) was performed, in which the ST-segment elevations were noted in the precordial leads (Fig. 1). The echocardiographic examination showed no significant deviations from the norm with an ejection fraction (EF) of 67%. Laboratory tests revealed significantly increased cardiac enzymes with troponin I levels 340× above the norm range, troponin T 80× above the norm range, creatine kinase-MB mass concentration 28× above the norm range and elevated levels of aspartate aminotransferase. Urine multitest confirmed the presence of tetrahydrocannabinol

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%