2005
DOI: 10.1016/j.biocel.2005.03.013
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Muscle wasting in cardiac cachexia

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Cited by 87 publications
(56 citation statements)
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References 85 publications
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“…In contrast, using a crossover design, short-term testosterone administration improved subjective and objective measures of exercise tolerance and metabolic factors in ten men with angina (Malkin et al 2004). Muscle atrophy secondary to systemic disorders such as cardiac failure, has no unifying aetiology, although a cytokine-mediated process associated with TNF-a, altered nitric oxide synthase expression and neuroendocrine alterations constitute plausible mechanisms (Strassburg et al 2005).…”
Section: Muscle Atrophy In Disuse Denervation and Medical Disordersmentioning
confidence: 97%
“…In contrast, using a crossover design, short-term testosterone administration improved subjective and objective measures of exercise tolerance and metabolic factors in ten men with angina (Malkin et al 2004). Muscle atrophy secondary to systemic disorders such as cardiac failure, has no unifying aetiology, although a cytokine-mediated process associated with TNF-a, altered nitric oxide synthase expression and neuroendocrine alterations constitute plausible mechanisms (Strassburg et al 2005).…”
Section: Muscle Atrophy In Disuse Denervation and Medical Disordersmentioning
confidence: 97%
“…Whether myostatin from the heart is also responsible for the insulin resistance or the fiber type switching (toward more slow fibers) reported to occur in heart failure was not yet examined, but both effects have been ascribed to myostatin (Fig. 4) (27,52,73,77). Interestingly, elimination of cardiac myostatin did only influence skeletal muscle mass after aortic constriction but not at baseline or after sham operation.…”
Section: Cardiac Myostatin In Health and Diseasementioning
confidence: 99%
“…Congestive heart failure is a leading cause of cardiovascular mortality and morbidity [11] and is associated with elevated circulating levels of angiotensin II [12] and muscle wasting, which is an important predictor of outcome in patients with this disease [1,13]. In rodents, angiotensin II infusion produced a marked reduction in body weight [14] and a reduction in circulating and skeletal muscle IGF-1.…”
Section: Heart Failure and Angiotensin IImentioning
confidence: 99%