Myocardial Infarction Without Atherosclerosis

Cheitlin, M.D.

doi:10.1001/jama.1975.03240210031015

Cited by 135 publications

(29 citation statements)

References 39 publications

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“…Furthermore, the coronary angiogram showed no signs of atherosclerosis. Many pathological causes of acute MI without atherosclerosis have been described [6]. In connection with acute leukemia the following four trigger mechanisms for MI and various electrocardiographic changes have been reported:…”

Section: Discussionmentioning

confidence: 99%

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Jachmann-Jahn

Cornely

Laufs

et al. 2001

Annals of Hematology

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A 42-year-old man was admitted with heavy retrosternal pain lasting 30 min. Electrocardiography showed typical signs of acute anterior myocardial infarction. The patient reported only attacks of coughing for a couple of days, and no serious diseases. The physical examination was normal. Laboratory tests showed a white blood cell count of 45/nl, platelet count of 58/nl, and hemoglobin of 14.4 g/dl. Blood chemistry showed elevated lactic dehydrogenase (413 U/l) but no elevation in creatine phosphokinase or glutamic-oxaloacetic transaminase. Therefore no thrombolysis was administered, but coronary angiography was performed. This showed a long-distance, subtotal thrombotic occlusion of the left anterior descending artery. After percutaneous transluminal coronary angioplasty and implantation of serial stents a normal perfusion of the artery was observed. The patient's blood and bone marrow films revealed acute myeloid leukemia FAB M2. Various conditions can cause a myocardial infarction in leukemias. We discuss the clinical management and the possible reasons for a subtotal thrombotic occlusion of the coronary artery.

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Section: Discussionmentioning

confidence: 99%

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Jachmann-Jahn

Cornely

Laufs

et al. 2001

Annals of Hematology

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“…First, they generally evaluate only post-MI patients, a group that may include relatively large numbers of individuals with nonatherosclerotic disease [27] who may be expected to have better prognoses than those with CAD [28]. Also, although there has been increasing interest in longterm follow-up for individuals with coronary risk factors [29], there has been little reported on long-term follow-up for those individuals with established coronary disease.…”

Section: Prognosis In Young Adults With Coronary Artery Diseasementioning

confidence: 99%

Premature coronary artery disease: Clinical risk factors and prognosis

Cole

Sperling

2004

Curr Atheroscler Rep

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Coronary artery disease (CAD) that becomes manifest in young adults can have devastating consequences. Additionally, the study of young patients with CAD may provide insight into the genetic basis of coronary disease. Over the past few years, our understanding of risk factors in this population has been expanded to include social, environmental, and emotional factors. The identification of genetic markers for disease is just beginning. Also, it has recently been shown that CAD in young adults has a poor long-term prognosis, meaning that clinicians caring for these patients must be aggressive in risk factor control.

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“…36], Consequently, patients with embolization are not necessarily 'cured', even after the excision of the primary tumor [37], Multiple organs. including spleen, kidney, bone, retina, skin, coronary and peripheral arteries can be affected by the emboli [15,29,36,38], Coro nary artery embolism leads to myocardial infarction [2,39]. Coronary arteriography revealed tumor embolus in the circumflex ar tery in 1 of our patients who sustained myo cardial infarction during the immediate post operative period.…”

Section: Discussionmentioning

confidence: 72%

Cardiac Myxoma – Manifestations and Management: Kuwait Experience and Review of the Literature

Uthaman

Salama²,

Hayat

et al. 1996

Med Principles Pract

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Over the past 14 years, 12 patients were diagnosed as having cardiac myxoma. All cases were sporadic, single, large, left atrial myxomas. These patients presented with nonspecific cardiac and noncardiac manifestations. The majority were misdiagnosed as valvar heart disease at initial evaluation, although echocardiography confirmed the diagnosis of myxoma in all. Prompt surgical excision of the tumor was carried out in 11 patients. Additionally, one of them underwent mitral valve repair. This patient died postoperatively due to a coronary embolism, and reoperation. One patient refused surgery. Of the 10 surviving patients who had operations, 9 remain asymptomatic and in sinus rhythm, while 1 has progressive mitral regurgitation and cardiac failure. Our experience indicates that prompt tumor excision under transesophageal echo guidance, shortly after diagnosis, yields excellent results with low morbidity and mortality.

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Myocardial Infarction Without Atherosclerosis

Cited by 135 publications

References 39 publications

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Premature coronary artery disease: Clinical risk factors and prognosis

Cardiac Myxoma – Manifestations and Management: Kuwait Experience and Review of the Literature

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Myocardial Infarction Without Atherosclerosis

Cited by 135 publications

References 39 publications

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Acute anterior myocardial infarction as first manifestation of acute myeloid leukemia

Premature coronary artery disease: Clinical risk factors and prognosis

Cardiac Myxoma &ndash; Manifestations and Management: Kuwait Experience and Review of the Literature

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Cardiac Myxoma – Manifestations and Management: Kuwait Experience and Review of the Literature