Physiotherapy and occupational therapy in the hypermobile adult

Keer, Rosemary; Butler, Katherine

doi:10.1016/b978-0-7020-3005-5.00013-6

Cited by 11 publications

(12 citation statements)

References 60 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Hypermobility syndromes are often marked by pain, nondermatomal paresthesia, decreased proprioception, impaired autonomic reflexes, and a predisposition toward neuropathies . In addition to joint hypermobility, other changes in connective tissue can involve other areas including the mitral valve, aorta, lens in the eye, and spinal dural ligaments .…”

Section: Causes Of Sacroiliac Joint Hypermobilitymentioning

confidence: 99%

“…7 Hypermobility syndromes are often marked by pain, nondermatomal paresthesia, decreased proprioception, impaired autonomic reflexes, and a predisposition toward neuropathies. 7,26 In addition to joint hypermobility, other changes in connective tissue can involve other areas including the mitral valve, aorta, lens in the eye, and spinal dural ligaments. 22,26 These familial conditions are typically more common in females, becoming symptomatic in late childhood and adolescence with symptoms of joint-related pain and stiffness, especially during long periods of standing or sitting.…”

Section: Genetic Joint Hypermobility Syndromesmentioning

confidence: 99%

“…7,26 In addition to joint hypermobility, other changes in connective tissue can involve other areas including the mitral valve, aorta, lens in the eye, and spinal dural ligaments. 22,26 These familial conditions are typically more common in females, becoming symptomatic in late childhood and adolescence with symptoms of joint-related pain and stiffness, especially during long periods of standing or sitting. 7,22 Affected individuals are prone to repetitivemotion overuse injuries due to their increased flexibility.…”

Section: Genetic Joint Hypermobility Syndromesmentioning

confidence: 99%

See 2 more Smart Citations

Sacroiliac Joint Hypermobility Biomechanics and What it Means for Health Care Providers and Patients

Enix

Mayer

2019

PM&R

View full text Add to dashboard Cite

Sacroiliac joint dysfunction is complex with numerous etiologies. Proper stabilization of the sacroiliac joint allows for effective transfer of loads between the trunk and the lower extremities during static and dynamic activities, while maintaining a freely nutating motion. A loss of integrity of the stabilizing soft‐tissue structures inhibits the ability to transmit axial loads and creates uneven stresses on the joint and surrounding tissues. Hypermobility of the sacroiliac joint can be caused by ligamentous instability or secondary to adaptive biomechanical changes and increased stresses affecting the joints of the pelvis. This article examines the current evidence related to the loss of stability on sacroiliac joint pain and dysfunction. A review of exercise goals for the hypermobile joint is included.

show abstract

Section: Causes Of Sacroiliac Joint Hypermobilitymentioning

confidence: 99%

Section: Genetic Joint Hypermobility Syndromesmentioning

confidence: 99%

Section: Genetic Joint Hypermobility Syndromesmentioning

confidence: 99%

See 1 more Smart Citation

Sacroiliac Joint Hypermobility Biomechanics and What it Means for Health Care Providers and Patients

Enix

Mayer

2019

PM&R

View full text Add to dashboard Cite

show abstract

“…Beighton's modification (Beighton & Horan, 1969) (Beighton P, Solomon L, 1973) of the Carter and Wilkinson scoring system (Carter C, 1964) is very popular for measuring generalised joint hypermobility (Nicholas JA, 1970b) (Alan J Hakim, Keer, & Grahame, 2010) (Lars Remvig, Jensen, & Ward, 2007)(L Remvig et al, 2007b) (Juul-Kristensen et al, 2007) (Ross & Grahame, 2011) (Simpson, 2006)(A.…”

Section: Limitationsmentioning

confidence: 99%

“…Hakim & Grahame, 2004) (Baum & Larsson, 2000)(Smits-Engelsman et al, 2011)(Alan J Hakim et al, 2010). However it has an inherent problem in that there is no accurate and specific cut off point to denote increased laxity, even in Beighton's own papers (Beighton & Horan, 1969) (Beighton P, Solomon L, 1973).…”

Section: Limitationsmentioning

confidence: 99%

Computed tomography scan evaluation for recurrent shoulder dislocations after sports injuries

Akhtar

Robinson

2010

Br J Sports Med

View full text Add to dashboard Cite

Shoulder dislocation is a common sporting injury. Recent studies suggest a higher rate of recurrence in the presence of glenoid rim fracture (bony Bankart lesion). The authors looked at the incidence of glenoid rim fracture, glenoid flattening and Hill-Sachs defect after recurrent shoulder dislocations. A prospective study was conducted in the Trauma and Orthopaedic Department of the Royal infirmary of Edinburgh from June 2008 to July 2009. All patients attending the specialist shoulder clinic after recurrent shoulder dislocations were evaluated with CT scan of their shoulder to look for glenoid rim fracture (bony Bankart lesion), glenoid flattening and Hill-Sachs defects. Prospective data were collected for 37 patients with a mean age of 25 years and a range of 14-40 years. 24 patients (71%) were involved in contact sports. The most common sport was football in 12 (50%) patients followed by rugby in 9 (37%) patients. Left shoulder was involved in 19 patients (51%). The CT scan showed glenoid fracture (bony Bankart lesion) in 11 patients (32%), glenoid flattening in 14 (41%) patients and Hill-Sachs defect in 29 (85%). The range of the defect was 11-45 mm. Four patients had glenoid flattening but no glenoid fracture. The type of instability was anterior in 33 (89%) patients and multidirectional instability in 4 (11%) patients. The number of dislocations range from 0 to 17 with a mean of 3 and the number of subluxations range from 0 to 35 with a mean of 4.5. The authors looked at the CT scan findings of glenoid and humeral head after recurrent shoulder dislocations in sports related injuries. Although glenoid rim fracture (bony Bankart lesion) is a risk factor for recurrence, glenoid flattening and large Hill-Sachs defects should be carefully evaluated with CT scan and appropriate stabilisation should be offered to prevent future problems.

show abstract