Primary ovarian insufficiency: a glimpse into the racial and socioeconomic disparities found within third-party reproduction

Wiltshire, Ashley; Ghidei, Luwam; Dawkins, Josette C.; Phillips, Kiwita; Licciardi, F.; Keefe, David L.

doi:10.1016/j.xfre.2021.09.004

Cited by 3 publications

(1 citation statement)

References 18 publications

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“…These two pathways are by no means exhaustive, and the complexity, various mechanisms, and epidemiologic underpinnings by which anti-Black racism negatively affects the health and well-being of Black women are being examined across disciplines and diverse fields of medicine. [10][11][12][13][14][15][16][17] Simultaneously, the use of Black "race" as a risk factor in medicine is being challenged across various health outcomes. [18][19][20][21] Accordingly, the intention of this article is to encourage a paradigm shift in our causal frameworks, one that embraces anti-racist thinking and praxis by recognizing the harm and limitedness of maintaining a race-based paradigm that labels Black "race" as the risk factor and instead names anti-Black racism as the root cause of these persistent disparities, thereby revealing important and highly treatable pathways (Fig.…”

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confidence: 99%

Why Causation Matters

Mayne,

Buckley,

Ghidei

2023

Obstetrics &Amp; Gynecology

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Although it is tempting to construe the correlation between Black “race” and higher rates of preterm birth as causal, this logic is flawed. Worse, the continued use of Black “race” as a risk factor for preterm birth is actively harmful. Using Black “race” as a risk factor suggests a causal relationship that does not exist and, critically, obscures what actually causes Black patients to be more vulnerable to poorer maternal and infant outcomes: anti-Black racism. Failing to name anti-Black racism as the root cause of Black patients' vulnerability conceals key pathways and tempts us to construe Black “race” as immutably related to higher rates of preterm birth. The result is that we overlook two highly treatable pathways—chronic stress and implicit bias—through which anti-Black racism negatively contributes to birth. Thus, clinicians may underuse important tools to reduce stress from racism and discrimination while missing opportunities to address implicit bias within their practices and institutions. Fortunately, researchers, physicians, clinicians, and medical staff can positively affect Black maternal and infant health by shifting our causal paradigm. By eliminating the use of Black “race” as a risk factor and naming anti-Black racism as the root cause of Black patients' vulnerability, we can practice anti-racist maternity care and take a critical step toward achieving birth equity.

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