Primary reticulum cell sarcoma of liver

Torres, Agustin; Bollozos, George D.

doi:10.1002/1097-0142(197106)27:6<1489::aid-cncr2820270632>3.0.co;2-d

Cited by 35 publications

(4 citation statements)

References 2 publications

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“…Later in 1971, Torres et al [19] described primary reticulum cell sarcoma of liver in cancer. Since then, cases and case series have been accumulated reporting the characteristics of primary hepatic lymphoma and stating that this disease is extremely rare with the absence of lymphoproliferative disease outside the liver.…”

Section: Classification Of Lymphomamentioning

confidence: 99%

Primary hepatic lymphoma: is it a disease entity?

Xiong¹,

Guan²

2017

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Section: Classification Of Lymphomamentioning

confidence: 99%

Primary hepatic lymphoma: is it a disease entity?

Xiong¹,

Guan²

2017

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“…In contrast, Torres (Torres, 1969;Torres and Bollozos, 1971), in reporting two cases of primary lymphoma of the liver, states that no previous case had been reported. A thorough review of the literature has revealed only one further case (Ata and Kamal, 1965).…”

Section: Discussionmentioning

confidence: 96%

A case of primary lymphoma of the liver.

Chambers¹,

O’Donoghue²,

Stansfeld³

1976

J Clin Pathol

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SYNOPSIS A case of primary malignant lymphoma of the liver is described. Presenting with epistaxis, he died in hepatic encephalopathy with an intractable bleeding post-bulbar duodenal ulcer.Although lymphomatous involvement of the liver is common in generalized lymphoma, lymphoma con-fined to the liver is exceptionally rare. Only three previous cases have been documented. The case described here is also unusual in being complicated by bleeding from a post-bulbar duodenal ulcer. Case reportA 55-year-old bank official presented at his local hospital with a severe epistaxis. He gave a six-week history of fatigue, anorexia, and loss of weight. His wife had noticed that at times he was confused and his speech was slurred.In 1945 he had developed malaria. This was treated routinely and there were no recurrences. Splenomegaly was noted at that time. There was no history of excess alcohol intake.On examination there was hepatosplenomegaly and he was admitted for further investigation. Over the next 48 hours he passed both fresh and altered blood per rectum. The prothrombin time and platelet count were normal, but his haemoglobin was 6-3 g/dl. Barium studies showed a post-bulbar duodenal ulcer and possible oesophageal varices. He was transfused 11 units of blood and transferred to St Bartholomew's Hospital.Examination on admission revealed a confused man with a marked flapping tremor and extrapyramidal rigidity. Foetor hepatis was present. There was no bruising of the skin nor petechial haemorrhages. Hepatosplenomegaly was present but there was no lymphadenopathy. None of the stigmata of chronic liver disease was present.Investigations at this time showed a post-transfusion haemoglobin of 13 g/dl, white cell count of 3800 cells/mm3, and a platelet count of 108 000/ mm3. Blood urea and serum electrolytes were normal.Received for publication 23 March 1976 Total protein was 4-4 g/dl, of which 2-3 was albumin. The bilirubin was 2.7 mg/dl, the alkaline phosphatase 575 IU/I, and the serum aspartate aminotransferase was 273 IU/I.It was thought that he had cirrhosis of the liver, and in view of the severity of his liver dysfunction no surgery was undertaken initially. Routine measures to combat hepatic encephalopathy were started but no improvement was noted. Six days after admission he again passed a melaena stool and became clinically shocked. Emergency endoscopy showed that the bleeding was from a post-bulbar duodenal ulcer. Oesophageal varices were not seen.At laparotomy later the same day the ulcer, measuring 2 x 1 cm, was located on the medial aspect of the second part of the duodenum. This was oversewn and a truncal vagotomy and Finney pyloroplasty were performed. The liver had a strange mottled appearance and a wedge biopsy was taken before the abdomen was closed.Postoperatively the patient failed to regain consciousness and continued to lose blood from the gastrointestinal tract. There was no evidence of disseminated intravascular coagulation. He failed to respond to transfusions of fresh blood and fresh frozen plasm...

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“…Fibroblastic reticulum cell sarcoma metastatic to the liver has been reported (Yaman et al 2009). In the older literature, several reports described so-called reticulum cell sarcoma of the liver (Torres and Bollozos 1971). Reticulum cells as they were classified in these times were slender and fusiform cells that were in close association with reticulum fibers and thought to be the producers of these fibers.…”

Section: Fbrc Tumors Of the Livermentioning

confidence: 99%

Tumors of the Dendritic Cell System

Zimmermann¹

2016

Tumors and Tumor-Like Lesions of the Hepatobiliary Tract

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Primary reticulum cell sarcoma of liver

Cited by 35 publications

References 2 publications

Primary hepatic lymphoma: is it a disease entity?

Primary hepatic lymphoma: is it a disease entity?

A case of primary lymphoma of the liver.

Tumors of the Dendritic Cell System

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