Proficiency testing for determination of formaldehyde in leather in China

Zhao, Yang; Guo, Dong; Huang, Zhiding; Chang, Wenkai; Qi, Xin; Li, Zenghe

doi:10.1016/j.trac.2016.06.014

Cited by 3 publications

(2 citation statements)

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“…Nevertheless, formaldehyde, a carcinogen that is harmful to human health and environment, was used as the aldehyde component in the preparation of MTA. 29,30 Consequently, the quest for new aldehyde components and further enhancement of the cationization degree are the key to the development of the amphoteric acrylic retanning agents prepared by Mannich reaction. In later studies, 31,32 glutaraldehyde, as an alternative for formaldehyde, was used to synthesize formaldehyde-free amphoteric retanning agents with certain assistant-dyeing properties.…”

Section: Introductionmentioning

confidence: 99%

Preparation and application of novel amphoteric acrylic retanning agents to improve dye absorption

Tian

Qiwu

et al. 2023

React. Chem. Eng.

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Section: Introductionmentioning

confidence: 99%

Preparation and application of novel amphoteric acrylic retanning agents to improve dye absorption

Tian

Qiwu

et al. 2023

React. Chem. Eng.

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“…Residual FA can be found in leather, flooring, and new cars. Although the release of FA from these materials has been significantly reduced by manufacturers in recent years, indoor concentrations in prefabricated houses have not decreased by the same amount [ 6 – 9 ]. In fact, FA has long been a topic of environmental policy discussions as an air-polluting substance which enters the body primarily through respiration.…”

Section: Introductionmentioning

confidence: 99%

The Effects of Formaldehyde on Cytochrome P450 Isoform Activity in Rats

Tang

Rong

et al. 2017

BioMed Research International

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Formaldehyde (FA) is an occupational and indoor pollutant. Long-term exposure to FA can irritate the respiratory mucosa, with potential carcinogenic effects on the airways. The effects of acute FA poisoning on the activities of CYP450 isoforms CYP1A2, CYP2C11, CYP2E1, and CYP3A2 were assessed by determining changes in the pharmacokinetic parameters of the probe drugs phenacetin, tolbutamide, chlorzoxazone, and testosterone, respectively. Rats were randomly divided into three groups: control, low FA dose (exposure to 110 ppm for 2 h for 3 days), and high FA dose (exposure to 220 ppm for 2 h for 3 days). A mixture of the four probe drugs was injected into rats and blood samples were taken at a series of time points. Plasma concentrations of the probe drugs were measured by HPLC. The pharmacokinetic parameters t1/2, AUC(0−t), and Cmax of tolbutamide, chlorzoxazone, and testosterone increased significantly in the high dose versus control group (P < 0.05), whereas the CL of chlorzoxazone and testosterone decreased significantly (P < 0.05). However, t1/2, AUC(0−t), and Cmax of phenacetin decreased significantly (P < 0.05), whereas the CL of phenacetin increased significantly (P < 0.05) compared to controls. Thus, acute FA poisoning suppressed the activities of CYP2C11, CYP2E1, and CYP3A2 and induced the activity of CYP1A2 in rats. And the change of CYP450 activity caused by acute FA poisoning may be associated with FA potential carcinogenic effects on the airways.

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