Relationship between gender role, anger expression, thermal discomfort and sleep onset latency in women

Arb, Mariella von; Gompper, Britta; Meyer, Andrea; Stutz, Elisabeth Zemp; Orgül, Selim; Kräuchi, Kurt

doi:10.1186/1751-0759-3-11

Cited by 13 publications

(9 citation statements)

References 24 publications

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“…cold hands and feet, in a Swiss study (Kräuchi et al., 2008; Mozaffarieh et al., 2010). In a follow‐up study, von Arb et al. (2009) suggest that ‘stereotypic feminine gender socialization may play an important determinant for anger suppression, which subsequently can lead to thermal discomfort from cold extremities’.…”

Section: Discussionmentioning

confidence: 99%

Thermal comfort and gender: a literature review

2011

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We should no longer neglect the more rigorous requirements that females have for indoor thermal environments. Gender differences indicate that females have, on average, a greater need for individual temperature control and adaptive actions than males. The results of this review suggest that females should primarily be used as subjects when examining indoor thermal comfort requirements, as if females are satisfied it is highly probable that males are also satisfied.

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Section: Discussionmentioning

confidence: 99%

Thermal comfort and gender: a literature review

2011

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“…Another trigger factor is psychological stress [45]. The way our body responds to this stress seems to be individually different [46] and also gender dependent [47]. Subjects with PVD often respond with redistribution of BF.…”

Section: Vascular Dysregulationmentioning

confidence: 99%

“…In glaucoma patients with PVD, we often observed very fast progression of visual field defects during a major stress event, for example, in the context of a divorce. It has recently been shown that women who commonly suppress anger experience more thermal discomfort from cold extremities and have prolonged sleep onset latency [47]. Furthermore, PVD subjects should avoid extensive cold.…”

Section: Therapymentioning

confidence: 99%

The primary vascular dysregulation syndrome: implications for eye diseases

2013

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Vascular dysregulation refers to the regulation of blood flow that is not adapted to the needs of the respective tissue. We distinguish primary vascular dysregulation (PVD, formerly called vasospastic syndrome) and secondary vascular dysregulation (SVD). Subjects with PVD tend to have cold extremities, low blood pressure, reduced feeling of thirst, altered drug sensitivity, increased pain sensitivity, prolonged sleep onset time, altered gene expression in the lymphocytes, signs of oxidative stress, slightly increased endothelin-1 plasma level, low body mass index and often diffuse and fluctuating visual field defects. Coldness, emotional or mechanical stress and starving can provoke symptoms. Virtually all organs, particularly the eye, can be involved. In subjects with PVD, retinal vessels are stiffer and more irregular, and both neurovascular coupling and autoregulation capacity are reduced while retinal venous pressure is often increased. Subjects with PVD have increased risk for normal-tension glaucoma, optic nerve compartment syndrome, central serous choroidopathy, Susac syndrome, retinal artery and vein occlusions and anterior ischaemic neuropathy without atherosclerosis. Further characteristics are their weaker blood–brain and blood-retinal barriers and the higher prevalence of optic disc haemorrhages and activated astrocytes. Subjects with PVD tend to suffer more often from tinnitus, muscle cramps, migraine with aura and silent myocardial ischaemic and are at greater risk for altitude sickness. While the main cause of vascular dysregulation is vascular endotheliopathy, dysfunction of the autonomic nervous system is also involved. In contrast, SVD occurs in the context of other diseases such as multiple sclerosis, retrobulbar neuritis, rheumatoid arthritis, fibromyalgia and giant cell arteritis. Taking into consideration the high prevalence of PVD in the population and potentially linked pathologies, in the current article, the authors provide recommendations on how to effectively promote the field in order to create innovative diagnostic tools to predict the pathology and develop more efficient treatment approaches tailored to the person.

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“…That glaucoma is associated with psychosomatic components is supported by the observation that women showing FS signs (e.g. cold extremities, prolonged sleep onset latency) have a tendency to suppress anger because of stereotypic feminine gender socialization ( van Arb et al, 2009 ), and normal-tension glaucoma patients show significantly more complaints and emotional instability ( Erb et al, 1999 ).…”

Section: Neurological Influences In “Ocular” Diseasesmentioning

confidence: 99%

Residual vision activation and the brain-eye-vascular triad: Dysregulation, plasticity and restoration in low vision and blindness – a review

Sabel

Merabet

2018

RNN

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Vision loss due to ocular diseases such as glaucoma, optic neuropathy, macular degeneration, or diabetic retinopathy, are generally considered an exclusive affair of the retina and/or optic nerve. However, the brain, through multiple indirect influences, has also a major impact on functional visual impairment. Such indirect influences include intracerebral pressure, eye movements, top-down modulation (attention, cognition), and emotionally triggered stress hormone release affecting blood vessel dysregulation. Therefore, vision loss should be viewed as the result of multiple interactions within a “brain-eye-vascular triad”, and several eye diseases may also be considered as brain diseases in disguise. While the brain is part of the problem, it can also be part of the solution. Neuronal networks of the brain can “amplify” residual vision through neuroplasticity changes of local and global functional connectivity by activating, modulating and strengthening residual visual signals. The activation of residual vision can be achieved by different means such as vision restoration training, non-invasive brain stimulation, or blood flow enhancing medications. Modulating brain functional networks and improving vascular regulation may offer new opportunities to recover or restore low vision by increasing visual field size, visual acuity and overall functional vision. Hence, neuroscience offers new insights to better understand vision loss, and modulating brain and vascular function is a promising source for new opportunities to activate residual vision to achieve restoration and recovery to improve quality of live in patients suffering from low vision.

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Relationship between gender role, anger expression, thermal discomfort and sleep onset latency in women

Cited by 13 publications

References 24 publications

Thermal comfort and gender: a literature review

Thermal comfort and gender: a literature review

The primary vascular dysregulation syndrome: implications for eye diseases

Residual vision activation and the brain-eye-vascular triad: Dysregulation, plasticity and restoration in low vision and blindness – a review

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