Reported occupational respiratory diseases in three spanish regions

Abstract

The compulsory scheme for reporting occupational respiratory diseases is seriously under-reporting in the three Spanish regions studied. Our voluntary surveillance program, which showed considerable differences in the characteristics and incidence of these diseases among the regions, appears to be more effective.

Twenty years of SHIELD: decreasing incidence of occupational asthma in the West Midlands, UK?

“…Other surveillance programmes have identified that cleaning workers account for a significant proportion of all work-related asthma. While the agents themselves are felt to be the cause, evidence exists for only a few cleaning products, with benzalkonium chloride, hypochlorite salts, ammonia and hydrochloric acid frequently implicated 39 40…”

A workforce-based study of occupational exposures and asthma symptoms in cleaning workers

“…Surveillance programs in different countries have shown that cleaning workers account for a considerable proportion of all work-related asthma cases, including both new-onset and work-aggravated asthma. Although cleaning products in general terms were considered the cause of asthma in many reported cases, only in very few instances was a specific cleaning product identified as causing asthma onset 10 11. Cleaning jobs may involve exposures related to the type of location where the cleaning is performed,12 but chemical cleaning products typically comprise the main exposure among cleaners.…”

Human Mesenchymal Stem Cells Resolve Airway Inflammation, Hyperreactivity, and Histopathology in a Mouse Model of Occupational Asthma

“…Persulfate salts used in hairdressing and cosmetics are well-known etiological agents involved in OA. These chemicals may be responsible for up to 5.8% of all cases of OA [2], representing the second, third, or fourth leading cause of OA according to different reports [2,3]. The mechanism by which persulfate salts cause OA is not well known, although based on clinical studies most authors support an immunologic mechanism [2,4].…”