RHD gene polymorphisms in alloimmunized RhD-negative individuals with high rate of racial admixture

It's time to phase inRHDgenotyping for patients with a serologic weakDphenotype

“…In response to the findings of the CAP TMRC survey, AABB and CAP convened a Work Group on RHD Genotyping and charged it with developing recommendations to clarify clinical issues related to RhD typing in persons with a serologic weak D phenotype. As an initial step for formulating recommendations, the Work Group reviewed the current state of molecular science of RHD , including more than 140 publications covering background; D variants with anti‐D; molecular basis of serologic weak D phenotypes; reviews, editorials, and commentaries; technical resources; and standards and guidelines . This commentary summarizes the proceedings and recommendations of the Work Group.…”

Rafting technique without bone grafting in reverse Hill-Sachs lesions

“…Biomechanical characteristics and high resistance to local depression loads for raft screw constructs and their clinical outcomes have been shown for split depression fractures of the tibial plateau. [13,14] Banerjee et.al. [24] reported satisfactory results with underpinning raft technique for reverse Hill-Sachs lesions.…”

“…[11,12] This technique is described especially for depressed tibial plateau fractures. [13] ÖZ Bu çalışmada, sağ tarafta eklem yüzeyinin %25-50'si ve sol tarafta %50'si kadar ters Hill-Sachs lezyonu olan çift taraflı bir posterior omuz instabilitesi olgusu bildirildi. Defektler tüberkulum minus osteotomisi sonrası eklem yüzeyinin yükseltilmesi ve greft kullanılmadan raft vidaları ile desteklenmesi yoluyla anatomik olarak yeniden yapılandırıldı.…”

Prospective Evaluation of a Transfusion Policy of RhD-Positive Red Blood Cells into DEL Patients in China

“…RHD (del Ex8) and RHD (del Ex9) alleles are classified as long deletions of the RHD gene [26,28], and RHD (del Ex9) is now determined to be nonexistent but resulted from a misinterpretation of data on RHD (1,227G>A) [12,27]. RHD-CE(4-9)-D belongs to RHD-CE-D hybrid gene [14,30,31,32]. Körmöczi and colleagues [9] proposed that DEL phenotypes should be divided into two subtypes: complete types where the majority of epDs are conserved such as RHD (1,227G>A) and partial D-like variants with characteristic epD loss caused by either RHD-CE-D hybrid genes or RHD point mutations RHD (IVS3 + 1G>A) affecting extracellular RhD loops.…”