2024
DOI: 10.14336/ad.2024.0309
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ROS-Induced Endothelial Dysfunction in the Pathogenesis of Atherosclerosis

Abstract: Various signaling pathways are regulated by reactive oxygen species (ROS), which are radical oxygen intermediates under normal physiological conditions. However, when the buffering capacity of antioxidant enzymes is exceeded by the accumulation of ROS, oxidative stress, and endothelial cell dysfunction occur, which have been recognized as key contributors to the development of atherosclerosis. In this review, an overview is provided on mechanisms underlying ROS generation in endothelial cells and the involved … Show more

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