Severe hypocalcemia mimicking ST-segment elevation acute myocardial infarction

Kukla, Piotr; Kulik, Monika; Jastrzębski, Marek; Bryniarski, Leszek; Czarnecka, Danuta; Baranchuk, Adrián

doi:10.1111/anec.12401

Cited by 4 publications

(3 citation statements)

References 10 publications

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“…As it is known, the duration of the ST segment is inversely proportional to the plasmatic calcium concentration [ 3 ]. Consistent with that, corrected QT interval was prolonged in most of the patients, except for the one described by Kukla et al [ 4 ], who presented with a shortened QT. Authors suggested the hypothesis of a coronary artery spasm as a possible cause.…”

supporting

confidence: 84%

Severe hypocalcemia mimicking acute ST-segment elevation myocardial infarction: Paradigmatic case and review of literature

Peiro

Cerdán²,

Diarte³

et al. 2022

Cardiol J.

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supporting

confidence: 84%

Severe hypocalcemia mimicking acute ST-segment elevation myocardial infarction: Paradigmatic case and review of literature

Peiro

Cerdán²,

Diarte³

et al. 2022

Cardiol J.

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“…22 La traducción electrocardiográfica más común de hipocalcemia es la prolongación del intervalo QT (Figura 5) como resultado del alargamiento del segmento ST, ocasionalmente, la hipocalcemia puede manifestarse con elevación del segmento ST, simulando un evento isquémico a nivel miocárdico. 23 Hipomagnesemia. El magnesio es un componente esencial de las estructuras del ácido desoxirribonucleico (ADN) y ácido ribonucleico (ARN) a nivel celular.…”

Section: )unclassified

Quemadura química por ácido fluorhídrico: mecanismos fisiopatológicos de la lesión

Escamilla¹,

Zúñiga²,

Coyotzin³

et al. 2022

Medicina Crítica

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Las quemaduras por exposición a químicos son una forma de lesión poco frecuente; sin embargo, los efectos secundarios se pueden considerar catastróficos a corto, mediano y largo plazo. El ácido fluorhídrico es una sustancia química perteneciente a los ácidos, tiene aplicación industrial y doméstica, y se encuentra en diferentes concentraciones. El fluoruro de hidrógeno está formado por un ion hidrógeno y un ion fluoruro, que al entrar en contacto con el agua adquiere su estado ácido, característica que le confiere mayor lesión de tejidos al entrar en contacto con ellos. Reportes describen lesiones de 1% de superficie corporal quemada, en una concentración al 50% capaz de generar toxicidad local y sistémica, que deriva en la muerte en cuestión de horas si no recibe un manejo oportuno encaminado a frenar el mecanismo de lesión, el cual se asocia a disminución de electrolitos calcio y magnesio, así como elevación de potasio, no sin dejar de lado la lesión corrosiva a nivel local que condiciona la aparición de necrosis licuefactiva. El objetivo de este trabajo es dar a conocer al personal médico y paramédico el mecanismo de lesión del ácido fluorhídrico, sus implicaciones clínicas y las alternativas terapéuticas.

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“…Although it is uncommon, hypocalcemia can cause ST-segment elevation [75]. After searching the PubMed and Google Scholar databases for hypocalcemia mimicking acute STEMI, many cases were found [75][76][77][78][79].…”

Section: Hypocalcemiamentioning

confidence: 99%

ST-Segment Elevation in Conditions of Non-cardiovascular Origin Mimicking an Acute Myocardial Infarction: A Narrative Review

Khurana¹,

Ranjan²

2022

Cureus

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The most widespread presenting ailments among patients visiting the emergency department are chest pain and shortness of breath. These symptoms lead any doctor to a probable diagnosis of myocardial infarction (MI). Detailed patient history, testing of blood samples for cardiac biomarkers that are indicative of cardiovascular necrosis, ultrasound methods, electrocardiography, and coronary computed tomography (CT) could all be beneficial to support the diagnosis. Out of these, electrocardiography is the most important and commonly done investigation in the emergency departments for patients presenting with chest pain and shortness of breath. However, interpreting these patients' electrocardiograms (ECGs) may be a matter of concern and worry. T wave and ST-segment changes are often of interest in the early signs of myocardial ischemia. Despite its incredible sensitivity, ST-segment deviation (elevated or depressed) has a low specificity because it can be seen in a variety of other cardiac and non-cardiac diseases. When ST-segment anomalies are identified, clinicians must consider many additional characteristics (such as risk factors, symptoms, and anamnesis), as well as all other possible diagnoses.All of these scenarios of patients presenting in the emergency department with chest discomfort and shortness of breath showing ST-segment abnormalities can leave a healthcare professional wondering whether to start treatment for acute myocardial infarction, through either the administration of a fibrinolytic agent, exposing patients to both the benefits and risks of fibrinolysis, or invasive coronary angiography. An astute physician may be able to recognize fabricated differential diagnosis mimicking STsegment elevation myocardial infarction (STEMI) in some situations. Failure to recognize these imposters can result in inefficient resource utilization, which can expose patients to unjustified risk and increased rather than decreased death and morbidity. Since the danger of cerebral hemorrhage from blood thinners is significant, in patient-care scenarios, in order to rule out percutaneous coronary intervention (PCI), a thorough assessment of the ECG is essential to consider diseases other than acute myocardial infarction, especially the ones that are non-cardiac in origin. The goal of this narrative review is to give an overview of the significant disorders that are non-cardiac in origin that can mimic an ST-segment elevation myocardial infarction (STEMI).

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Severe hypocalcemia mimicking ST-segment elevation acute myocardial infarction

Cited by 4 publications

References 10 publications

Severe hypocalcemia mimicking acute ST-segment elevation myocardial infarction: Paradigmatic case and review of literature

Severe hypocalcemia mimicking acute ST-segment elevation myocardial infarction: Paradigmatic case and review of literature

Quemadura química por ácido fluorhídrico: mecanismos fisiopatológicos de la lesión

ST-Segment Elevation in Conditions of Non-cardiovascular Origin Mimicking an Acute Myocardial Infarction: A Narrative Review

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