Small vessel vasculitis caused by hepatitis B virus immune complexes *1, *2Small vessel vasculitis and HBsAg

Gower, Richard; Sausker, William F.; Köhler, Peter F.; Thorne, G.E.; McIntosh, Rawle M.

doi:10.1016/0091-6749(78)90211-7

Cited by 100 publications

(27 citation statements)

References 18 publications

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“…HBsAg, IgM and C3 were demonstrable in the arteriolar walls of the skin. The results support the concept that complement activating immunecomplexes containing HBsAg and IgM anti HBs play a role in the pathogenesis of vasculitis as described here 6,7 . Like chronic persistent hepatitis B , acute viral hepatitis B may also be complicated with HSP 8,9 .…”

Section: Discussionsupporting

confidence: 78%

Henoch-Schönlein Purpura as a Presentation of Chronic Hepatitis B Infection

Begum¹,

Haque²,

Trisha³

et al. 2014

J. Medicine

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Henoch-Schönlein Purpura (HSP) is characterized by presence of classical triad of palpable purpura, arthritis and abdominal pain 1. HSP may be triggered by bacterial and viral infections, eg. Hepatitis B viral infection. Chronic hepatitis B infection may present with extra hepatic menifestations, vasculitis is an important one and poly arteritis nodosa is more commonly seen. A 30 years old lady presented with abdominal pain, palpable rashes over trunk, lower limb, which were consistent with HSP along with joint pain more on knees and ankles. She was diagnosed as a case of chronic hepatitis. HSP is not very uncommon among patients attending hospitals of Bangladesh, but its relation with Hepatitis B virus (HBV) or any other infective agent is not always searched. In our case HSP presented as an extra hepatic manifestation of chronic hepatitis B infection. The extra hepatic manifestations of chronic hepatitis B are associated with deposition of circulating HBV Ag-Ab immune complexes.DOI: http://dx.doi.org/10.3329/jom.v14i2.19692 J Medicine 2013, 14(2): 210-212

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Section: Discussionsupporting

confidence: 78%

Henoch-Schönlein Purpura as a Presentation of Chronic Hepatitis B Infection

Begum¹,

Haque²,

Trisha³

et al. 2014

J. Medicine

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“…Viral antigens have been detected in vessel walls in several forms of vasculitis (4,5). For example, immune complex deposition in the cutaneous microvasculature after herpes simplex virus (HSV)1 infection has been implicated in the development of erythema multiforme in some patients (6).…”

Section: Introductionmentioning

confidence: 99%

Fc and C3 Receptors Induced by Herpes Simplex Virus on Cultured Human Endothelial Cells

Cines¹,

Lyss²,

Bina³

et al. 1982

J. Clin. Invest.

134

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“…Viruses may also activate neutrophils indirectly by stimulating release of cytokines such as interferon-y from mononuclear cells (7). Viral immune complexes that may occur in measles, rubella, and hepatitis B will initiate oxidative burst activity and arachidonic acid metabolism in neutrophils (8,9).…”

mentioning

confidence: 99%