Studies in the Hemodynamics Following Supradiaphragmatic Constriction of the Inferior Vena Cava*

Seitchik, Murray W.; Poll, Marvin; Rosenthal, William S.; Baronofsky, Ivan D.

doi:10.1097/00000658-196101000-00008

Cited by 5 publications

(2 citation statements)

References 2 publications

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“…Also based on the pressures obtained with this cross-validation approach, we calculated the sensitivity, specificity and the accuracy of identifying PH in these canines choosing a threshold value of 16 mmHg, because this represents the clinical scenario of HVPG values corresponding to moderate through severe PH cases. For canines, this value of 16 mmHg was selected based on data presented in literature (Seitchik et al 1961, Yamana et al 1983, Palmaz et al 1986, Sugita et al 1987, Chen et al 2009, Jin et al 2010, Buob et al 2011). …”

Section: Methodsmentioning

confidence: 99%

Investigating the Efficacy of Subharmonic Aided Pressure Estimation for Portal Vein Pressures and Portal Hypertension Monitoring

Dave¹,

Halldorsdottir²,

Eisenbrey³

et al. 2012

Ultrasound in Medicine & Biology

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The efficacy of using subharmonic emissions from Sonazoid microbubbles (GE Healthcare, Oslo, Norway) to track portal vein pressures and pressure changes was investigated in 14 canines using either slow- or high-flow models of portal hypertension (PH). A modified Logiq 9 scanner (GE Healthcare, Milwaukee, WI) operating in subharmonic mode (ftransmit:2.5MHz, freceive:1.25MHz) was used to collect RF data at 10-40% incident acoustic power levels with 2-4 transmit cycles (in triplicate), before and after inducing PH. A pressure catheter (Millar Instruments, Inc., Houston, TX) provided reference portal vein pressures. At optimum insonification, subharmonic signal amplitude changes correlated with portal vein pressure changes; r ranged from -0.82 to -0.94 and from -0.70 to -0.73 for PH models considered separately or together, respectively. The subharmonic signal amplitudes correlated with absolute portal vein pressures (r: -0.71 to -0.79). Statistically significant differences between subharmonic amplitudes, before and after inducing PH, were noted (p≤0.01). Portal vein pressures estimated using SHAPE did not reveal significant differences (p>0.05) with respect to the pressures obtained using the Millar pressure catheter. Subharmonic aided pressure estimation may be useful clinically for portal vein pressure monitoring.

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Section: Methodsmentioning

confidence: 99%

Investigating the Efficacy of Subharmonic Aided Pressure Estimation for Portal Vein Pressures and Portal Hypertension Monitoring

Dave¹,

Halldorsdottir²,

Eisenbrey³

et al. 2012

Ultrasound in Medicine & Biology

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“…Most of the existing animal models of subdiaphragmatic congestion do not meet the criteria to be a clinically relevant animal model of isolated congestion (Table 2). First, the exact technique to induce congestion [44, 45, 47, 48, 55, 56], the anatomical location of where the constriction is applied [44, 46, 49, 52, 53, 59–68], and the degree of constriction were not always properly described [46, 47, 50, 52, 54, 56, 65, 66, 68, 69]. Second, it was not always clear whether venous congestion was actually induced, since hemodynamic or echocardiographic measurements were not performed in these studies [46–52, 54–56, 58, 59, 69].…”

Section: Animal Models Of Isolated Systemic Congestionmentioning

confidence: 99%

Current animal models for the study of congestion in heart failure: an overview

et al. 2019

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Congestion (i.e., backward failure) is an important culprit mechanism driving disease progression in heart failure. Nevertheless, congestion remains often underappreciated and clinicians underestimate the importance of congestion on the pathophysiology of decompensation in heart failure. In patients, it is however difficult to study how isolated congestion contributes to organ dysfunction, since heart failure and chronic kidney disease very often coexist in the so-called cardiorenal syndrome. Here, we review the existing relevant and suitable backward heart failure animal models to induce congestion, induced in the left-(i.e., myocardial infarction, rapid ventricular pacing) or right-sided heart (i.e., aorta-caval shunt, mitral valve regurgitation, and monocrotaline), and more specific animal models of congestion, induced by saline infusion or inferior vena cava constriction. Next, we examine critically how representative they are for the clinical situation. After all, a relevant animal model of isolated congestion offers the unique possibility of studying the effects of congestion in heart failure and the cardiorenal syndrome, separately from forward failure (i.e., impaired cardiac output). In this respect, new treatment options can be discovered.

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Anesthetic and Hemodynamic Considerations of Inferior Vena Cava Surgery

Eyraud

Lepère

2017

Surgery of the Inferior Vena Cava

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Studies in the Hemodynamics Following Supradiaphragmatic Constriction of the Inferior Vena Cava*

Cited by 5 publications

References 2 publications

Investigating the Efficacy of Subharmonic Aided Pressure Estimation for Portal Vein Pressures and Portal Hypertension Monitoring

Investigating the Efficacy of Subharmonic Aided Pressure Estimation for Portal Vein Pressures and Portal Hypertension Monitoring

Current animal models for the study of congestion in heart failure: an overview

Anesthetic and Hemodynamic Considerations of Inferior Vena Cava Surgery

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