Surface Cooling (20 °C) and Circulatory Arrest in Infants Undergoing Cardiac Surgery

Lamberti, John J.; Lin, Chung-Yuan; Cutilletta, Anthony F.; Anagnostopoulos, Constantine E.; Arcilla, René A.; Replogle, Robert L.

doi:10.1001/archsurg.1978.01370190044007

Cited by 9 publications

(1 citation statement)

References 11 publications

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“…is now the procedure of the choice in many centres. Several studies [2][3][4] have demonstrated a lower operative mortality for primary closure when compared with a two-staged approach with initial pulmonary artery banding. As outlined in an earlier reports 5 , direct surgery of a large VSD has been recommended for infants demonstrating intractable congestive heart failure, persistent or recurrent pneumonia, marked growth retardation and severe pulmonary hypertension (that is, pulmonary arterial pressure greater than 75% of the systemic pressure).…”

Section: Bas J Surg March 9 2003mentioning

confidence: 99%

Hemodynamic Changes after Surgical Closure of Ventricular Septal Defect.

Haweleh¹

2003

Basrah Journal of Surgery

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This is a prospective study that was carried out during the period between January 1990 to January 2000, to assess the hemodynamic changes and outcome after surgical closure of ventricular septal defect (VSD). Out of 150 patients who underwent surgical closure of VSD, 52 patients had follow up cardiac catheterisation. The main indications for recatheterisation were: persistent respiratory symptoms, clinical or echocardiographic signs of residual leak across the VSD, and improper weight gain. The time interval between recath and surgery was ranging from 3 to 6 months. Complete closure was achieved in 35 patients out of 52 (76%) who had been recatheterized, persistent severe pulmonary hypertension was noticed in one patient who underwent atrial septostomy, 4(8%) patients had significant leak across the defect and high pulmonary hypertension needed a second operation, 12(23%) patients had small residual leak. The mean pulmonary arterial pressure has dropped from 60mmHg to 27 mmHg. Two patients had complete heart block needed an insertion of permanent pacemaker. The hospital mortality of the entire group was 2% and there was no late mortality. In conclusion, hemodynamic changes following the surgical closure of the ventricular septal defect had shown further reduction in the pulmonary pressure and pulmonary vascular resistance as early as 3 months in most patients, rarely some patients needed another attempt of closure of the residual VSD leak. Surgical atrial septostomy might be needed in patients with persistent severe pulmonary hypertension to decompress the right side of the heart.

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Section: Bas J Surg March 9 2003mentioning

confidence: 99%

Hemodynamic Changes after Surgical Closure of Ventricular Septal Defect.

Haweleh¹

2003

Basrah Journal of Surgery

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Incidence and Prognosis of Seizures in Infants After Cardiac Surgery With Profound Hypothermia and Circulatory Arrest

Ehyai¹

1984

JAMA

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Fifteen of 165 infants and young children who underwent surgical correction of congenital cardiac defects using profound hypothermic and circulatory arrest experienced generalized or focal seizures postoperatively. The cause of the seizures was unexplained in ten. Among these ten, the onset of seizures was 25 to 48 hours after surgery. With appropriate treatment, all had complete seizure control by the third postoperative day. During 11 to 54 months (mean, 35.6 months) of follow-up, no further seizures occurred and none had neurological abnormalities. Long-term anticonvulsant therapy was not required for any of the children. There was no correlation between the type of cardiac abnormality (cyanotic v acyanotic) or the duration of hypothermic circulatory arrest and the development of seizures. Unexplained seizures following cardiac surgery with hypothermia and circulatory arrest are not a sign of permanent brain damage and do not detract from the use of this technique for early definitive repair of congenital heart defects.

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