Suspicion de mycotoxicose provoquée par les trichothècènes chez les poulets de chair

Renault, Lucien; Goujet, Martine; Monin, Anne; Boutin, G.; Palisse, M.; Alamagny, A.

doi:10.4267/2042/65459

Cited by 4 publications

(2 citation statements)

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“…Avian fusariotoxicosis is considered to be caused by trichothecenes because field and experimental forms of the disease involve ulceration of the oral mucosa. T-2 toxin was a cause of avian fusariotoxicosis in the United States (Wyatt et al, 1972), Canada (Greenway and Puls, 1976) and France (Renault et al, 1979). Avian fusariotoxicosis occurred in Russia in association with human alimentary toxic aleukia (Kurmanov and Novacky, 1978).…”

Section: Introductionmentioning

confidence: 99%

Experimental trichothecene mycotoxicosis produced in broiler chickens byFusarium sporotrichiellavar.sporotrichioides

et al. 1982

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SUMMARYFusarium sporotrichiella var. sporotrichioides (Bilay), cultured on . sterilised popcorn at 23°C and then at 8°C, 16°C and 23°C and fed as 50% of the diet, was lethal to 7-day-old male broiler chickens. The 8°C culture, containing T-2 toxin at 50 parts per million (ppm) and neosolaniol at 5 ppm, was given as whole culture at dietary concentrations of 10%, 5%, 1% and 0% for 17 days and 1% for 42 days. Half the chickens that were fed the 10% diet died during the 17 days (5 ppm T-2 toxin and 0.5 ppm neosolaniol). The corresponding daily dose was 0.24 mg T-2 toxin and 0.02 mg neosolaniol/kg body weight/day. The chickens that died were dehydrated, had necrosis and depletion of lymphoid and haematopoietic tissues and necrosis of the hepatobiliary system, gastroenteric mucosa, feather epidermis and renal tubular epithelium. The survivors had anaemia, reduction of weight gain and transiently altered righting reflex. The comb and beak were pale yellow and the feather barbs were dishevelled. Survivors also had atrophied lymphoid tissues, reduced haematopoietic cellularity in the bone marrow, necrosis of oral and crop mucosa, vacuolated hepatocytes, hyperplastic bile ductules, and reduction of the thyroid follicular diameter.

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Section: Introductionmentioning

confidence: 99%

Experimental trichothecene mycotoxicosis produced in broiler chickens byFusarium sporotrichiellavar.sporotrichioides

et al. 1982

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“…T-2 toxin is a cause of fusariotoxicosis of chickens (Renault et al, 1979), pigeons (Wyatt et al, 1972), and geese and ducks (Greenway and Puls, 1976). Diacetoxyscirpenol is similar to T-2 toxin except at the carbon 8 moiety.…”

Section: Introductionmentioning

confidence: 99%

Mycotoxicosis produced in broiler chickens by multiple doses of either t‐2 toxin or diacetoxyscirpenol

et al. 1982

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SUMMARYT-2 toxin, a 12, 13-epoxytrichothecene mycotoxin, was given by crop gavage to 7-day-old male broiler chickens as 14 daily doses of 1.5, 2.0, 2.5, and 3.0 mg/kg body weight/day. Diacetoxyscirpenol, also an epoxytrichothecene, was given at doses of 2.5, 3.0,and 3.5 mg/kg/day. Some chickens became dehydrated and emaciated and died. In survivors, body weight and haematocrit were reduced, the feathers were malformed, and the beak and legs were pale yellow. At necropsy, the lymphoid organs were atrophic, bone marrow was pale red or yellow, the liver was discoloured yellow, and the crop mucosa was ulcerated.Microscopic lesions included necrosis and cell depletion in lymphocytic and haematopoietic tissues, and necrosis of hepatocytes, bile ductule epithelium, enteric mucosa, and germinal regions of feather barbs. Other hepatic lesions were fatty change of hepatocytes and hyperplasia of bile ductules. Thyroid follicles were small, contained pale colloid and had tall epithelial cells. T-2 toxin was more detrimental than diacetoxyscirpenol to lymphocytic and haematopoietic tissues.

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Screening fusarium strains isolated from overwintered Canadian grains for trichothecenes

1980

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A survey of 38 samples of Canadian overwintered grains showed that 14 (37%) contained viable Fusarium. Of a total of 38 Fusarium isolates, cultured on autoclaved corn, 20 (from 7 grain samples) showed toxicity to brine shrimp larvae and 12 (from 5 samples) produced levels of trichothecenes detectable by thin layer chromatography. The principal trichothecene found was T-2 toxin, produced by 10 strains and accompanied in half of these by neosolaniol; some of these strains were identified as F. sporotrichioides Sherbakoff. Two strains of F. poae (Peck) Wollenw. formed small amounts of diacetoxyscirpenol. T-2 toxin was the most toxic of 8 trichothecenes tested on brine shrimp larvae; the wide range of toxicities limits the usefulness of this bioassay as a general screening method for trichothecenes.

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Suspicion de mycotoxicose provoquée par les trichothècènes chez les poulets de chair

Cited by 4 publications

References 0 publications

Experimental trichothecene mycotoxicosis produced in broiler chickens byFusarium sporotrichiellavar.sporotrichioides

Experimental trichothecene mycotoxicosis produced in broiler chickens byFusarium sporotrichiellavar.sporotrichioides

Mycotoxicosis produced in broiler chickens by multiple doses of either t‐2 toxin or diacetoxyscirpenol

Screening fusarium strains isolated from overwintered Canadian grains for trichothecenes

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