Takotsubo Cardiomyopathy as a Consequence of Myocardial Infarction. It is Possible?

Muksinova, M. D.; Shilova, Alexandra; Gilyarov, M. Yu.; Константинова, Е. В.; Nesterov, A. P.; Udovichenko, A. E.; Свет, А. В.

doi:10.18087/cardio.2017.12.10071

Cited by 2 publications

(1 citation statement)

References 9 publications

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“…Of note is that CAD and ACS, diseases that need to be differentiated from TTS, are occasionally comorbidities of TTS, and may occasionally precipitate secondarily TTS [ 95 , 96 ]. In such cases of concurrence of ACS and TTS, management of a coronary occlusion in a patient with AMI and TTS should be undertaken as performed routinely in accordance with issued guidelines for AMI, including the employment of percutaneous coronary interventions (PCI) [ 97 ], or even coronary artery bypass graft (CABG) surgery.…”

Section: Current Therapy Of Acute Ttsmentioning

confidence: 99%

Takotsubo Cardiomyopathy: Current Treatment

Madias

2021

JCM

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Management of takotsubo syndrome (TTS) is currently empirical and supportive, via extrapolation of therapeutic principles worked out for other cardiovascular pathologies. Although it has been emphasized that such non-specific therapies for TTS are consequent to its still elusive pathophysiology, one wonders whether it does not necessarily follow that the absence of knowledge of TTS’ pathophysiological underpinnings should prevent us for searching, designing, or even finding, therapies efficacious for its management. Additionally, it is conceivable that therapy for TTS may be in response to pathophysiological/pathoanatomic/pathohistological consequences (e.g., “myocardial stunning/reperfusion injury”), common to both TTS and coronary artery disease, or other cardiovascular disorders). The present review outlines the whole range of management principles of TTS during its acute phase and at follow-up, including considerations pertaining to the recurrence of TTS, and commences with the idea that occasionally management of TTS should consist of mere observation along the “first do no harm” principle, while self-healing is under way. Finally, some new therapeutic hypotheses (i.e., large doses of insulin infusions in association with the employment of intravenous short- and ultrashort-acting β-blockers) are being entertained, based on previous extensive animal work and limited application in patients with neurogenic cardiomyopathy and TTS.

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Section: Current Therapy Of Acute Ttsmentioning

confidence: 99%

Takotsubo Cardiomyopathy: Current Treatment

Madias

2021

JCM

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Toxic Myocardial Injury in a Patient With Coronary Atherosclerosis, Caused by Acute Poisoning With Gaseous Chlorine

Alidzhanova¹,

Moiseeva²,

Пашаева³

et al. 2021

Neotložnaâ med. pomoŝʹ

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Acute chlorine gas poisoning leads to activation of the sympathetic nervous system and, as a consequence, dysfunction of the cardiovascular system (CVS). We report a clinical case of toxic myocardial injury with gaseous chlorine. In a man with coronary artery disease and polypathy, toxic myocardial injury mimicked acute coronary syndrome (ACS) and was accompanied by a large area of left ventricular microvascular dysfunction, which did not coincide with the areas of blood supply of altered coronary arteries; the dynamics of electrocardiographic changes resembled myocardial stunning in Takotsubo syndrome (TS). The effect of chlorine on CVS, features of clinical and instrumental diagnostics and differentiation of primary / secondary CT and ACS are discussed.

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Takotsubo Cardiomyopathy as a Consequence of Myocardial Infarction. It is Possible?

Cited by 2 publications

References 9 publications

Takotsubo Cardiomyopathy: Current Treatment

Takotsubo Cardiomyopathy: Current Treatment

Toxic Myocardial Injury in a Patient With Coronary Atherosclerosis, Caused by Acute Poisoning With Gaseous Chlorine

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