The effect of a nicotine patch on cigarette craving over the course of the day: results from two randomized clinical trials

Shiffman, Saul; Ferguson, Stuart G.

doi:10.1185/03007990802380341

Cited by 23 publications

(38 citation statements)

References 27 publications

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“…These investigations confirmed the anti-craving effect of nicotine patches (Bolt et al, 2012;Ferguson et al, 2006;Shiffman et al, 2000Shiffman and Ferguson, 2008), nicotine gum (O'Connell et al, 2007), bupropion Piper et al, 2008), naltrexone (Miranda et al, 2013;Tidey et al, 2008), varenicline (Gass et al, 2012), but also individualized cognitive-behavioral treatment (IATP) compared to traditional cognitive-behavioral treatment (CBT) (Litt et al, 2009). Heroin craving was found to be higher in patients with higher doses of methadone (De Vos et al, 1996).…”

Section: Intra-individual Variablessupporting

confidence: 75%

Ecological momentary assessment in the investigation of craving and substance use in daily life: A systematic review

Serre

Fatséas

Swendsen

et al. 2015

Drug and Alcohol Dependence

315

222

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Section: Intra-individual Variablessupporting

confidence: 75%

Ecological momentary assessment in the investigation of craving and substance use in daily life: A systematic review

Serre

Fatséas

Swendsen

et al. 2015

Drug and Alcohol Dependence

315

222

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“…It is interesting to note that nicotine patches had only a weak effect in attenuating craving in this study. This is in contrast with signifi cant craving suppression outcome observed with nicotine patch in previous studies (e.g., Shiffman & Ferguson, 2008 ;Tiffany, Cox, & Elash, 2000 ;Transdermal Nicotine Study Group, 1991 ). It is not clear what factors might have contributed to this discrepancy.…”

Section: Effects On Postquit Cravingcontrasting

confidence: 55%

DRD2 -related TaqIA polymorphism modulates motivation to smoke

Zuo

Gilbert²,

Riise³

et al. 2009

Nicotine &Amp; Tobacco Research

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Introduction: TaqIA polymorphism, a genetic variant associated with the expression level of dopamine D2 receptors in the brain, has been linked to various aspects of smoking behavior, including smoking prevalence, affective withdrawal symptoms, and smoking cessation outcome. However, its involvement in motivation to smoke cigarettes has not been elucidated. Methods:The present study examined the possible differences in self-reported reasons to smoke and craving for smoking in 160 smokers participating in a clinical trial.Results: Individuals with at least one A1 allele of the TaqIA polymorphism were more likely to report smoking for stimulating effects and to reduce negative affect compared with those lacking an A1 allele. The association of the A1 genotype with a higher probability and stronger motive to smoker to enhance cognitive functioning was evident in female but not in male smokers. Female A1 carriers also expected a greater likelihood of smoking for pleasure than those without an A1 allele. A1 subjects reported stronger craving for cigarettes during early days and the last phase of a 6-week abstinence period. Discussion:These results support the idea that dopaminergic transmission plays an important role in the neurobiological basis of reasons for smoking and that the TaqIA variant is one of the genetic factors underlying individual differences in these aspects. These fi ndings also have implications for improving treatment strategies to help individuals quit smoking by controlling their motivation to continue cigarette consumption.

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“…5 Individual variables related Hitsman et al, 2013;Shiffman & Ferguson, 2008), very few studies have explored which variables are associated with craving reductions. Those studies have mainly explored the influence of gender (Collins, Nair, & Komaroff, 2011;Unrod et al, 2013), nicotine dependence (Canterberry et al, 2013;Kober et al, 2010;Li et al, 2013) and…”

Section: Accepted Manuscriptmentioning

confidence: 99%