2015
DOI: 10.2174/1872208309666150615142218
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The Tendon Approximator Device in Traumatic Injuries

Abstract: The new nerve approximator device is cheap, feasible to use and reduces the time of tendon repair with sustained outcomes comparable to the conventional methods.

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“…Based on the evidence that several flavonoids are able to activate PPARγ [20][21][22][23] and that PPARγ is actively involved in the regulation of angiogenesis, [25] we proposed a hypothesis that morin might exert its anti-angiogenic effects by activating PPARγ . To verify this, we utilized HUVECs to explore the suppressive effect of morin on VEGF-induced endothelial cell migration and tube formation in vitro and treated HUVECs with PPARγ siRNA, GW9662 (PPARγ antagonist), and/or rosiglitazone (PPARγ agonist) to ascertain the involvement of PPARγ .…”
Section: Morin Inhibited Vegf-induced Endothelial Cell Migration and mentioning
confidence: 99%
“…Based on the evidence that several flavonoids are able to activate PPARγ [20][21][22][23] and that PPARγ is actively involved in the regulation of angiogenesis, [25] we proposed a hypothesis that morin might exert its anti-angiogenic effects by activating PPARγ . To verify this, we utilized HUVECs to explore the suppressive effect of morin on VEGF-induced endothelial cell migration and tube formation in vitro and treated HUVECs with PPARγ siRNA, GW9662 (PPARγ antagonist), and/or rosiglitazone (PPARγ agonist) to ascertain the involvement of PPARγ .…”
Section: Morin Inhibited Vegf-induced Endothelial Cell Migration and mentioning
confidence: 99%