Zhou, T.B., et al., Correction: All-Trans Retinoic Acid Treatment Is Associated with Prohibitin Expression in Renal Interstitial Fibrosis Rats. Int. J. Mol. Sci. 2012, 13, 2769-2782.

Abstract

The authors wish to change Figure 2 of the paper published in IJMS [1]. The positions of H1 and H2 in the previous article were reversed. These errors have been amended in an amended version of the manuscript, which is available from the International Journal of Molecular Sciences website. The authors and publisher apologize for the inconvenience. [...

A synthetic retinoic acid receptor agonist Am80 ameliorates renal fibrosis via inducing the production of alpha-1-acid glycoprotein

“…(UUO)-induced renal fibrosis model 8 , and retinoids have protective effects in numerous types of renal damage and inflammation including acute pyelonephritis 9 and glomerulosclerosis in adriamycin-induced nephropathy model 10,11 In addition, Chiba et al reported that atRA treatment reduced macrophage-dependent injury and fibrosis after an acute kidney injury (AKI), where atRA functioned to regulate macrophage activation 12 . These collective studies indicate that retinoids play a protective role in renal injury.…”