PI 698653) is an early maturity group IV (relative maturity 4.4) conventional soybean [Glycine max (L.

The conventional Maturity Group V soybean [Glycine max (L). Merr.] cultivar ‘S13‐1955C’ (Reg. no. CV‐540, PI 695317) was developed and released by the University of Missouri–Fisher Delta Research Center soybean breeding program in 2019. Plants of S13‐1955C have determinate growth habit, white flowers, tawny pubescence, tan pod wall, and seed with black hilum. It has high oil content (227.4 g kg−1) and broad disease resistance including soybean cyst nematode, southern root‐knot nematode, reniform nematode, sudden death syndrome, and frogeye leaf spot. S13‐1955C was tested in 177 environments across 13 states and averaged 101% of the check yield in Missouri and the southern states from 2014 to 2019.

This review summarizes the history and current state of the known genetic basis for soybean resistance to Soybean mosaic virus (SMV), and examines how the integration of molecular markers has been utilized in breeding for crop improvement. SVM causes yield loss and seed quality reduction in soybean based on the SMV strain and the host genotype. Understanding the molecular underpinnings of SMV–soybean interactions and the genes conferring resistance to SMV has been a focus of intense research interest for decades. Soybean reactions are classified into three main responses: resistant, necrotic, or susceptible. Significant progress has been achieved that has greatly increased the understanding of soybean germplasm diversity, differential reactions to SMV strains, genotype–strain interactions, genes/alleles conferring specific reactions, and interactions among resistance genes and alleles. Many studies that aimed to uncover the physical position of resistance genes have been published in recent decades, collectively proposing different candidate genes. The studies on SMV resistance loci revealed that the resistance genes are mainly distributed on three chromosomes. Resistance has been pyramided in various combinations for durable resistance to SMV strains. The causative genes are still elusive despite early successes in identifying resistance alleles in soybean; however, a gene at the Rsv4 locus has been well validated.