2 The growth of children with chronic paediatric disease

Preece, M A; Law, Caroline; Davies, P. S. W.

doi:10.1016/s0300-595x(86)80006-8

Cited by 41 publications

(24 citation statements)

References 92 publications

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“…It has been established since the 1980s that the children with BA tend to be lower in height than healthy children [35]. In the study carried out by Umlawska et al, 2013, which included 261 children with bronchial asthma, it was concluded that about 5% of the children with BA were very short in stature: their height was under the 5th percentile of the growth charts [48].…”

Section: Resultsmentioning

confidence: 99%

Review of different anthropometric and nutritional measurements in children with bronchial asthma

Lipsberga

Kažoka

2015

PoA

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The aims of this study were to describe and compare the recent literature data of anthropometric and nutritional measurements in children with bronchial asthma from the researches published in the last 10 years. 46 studies from the year 2003 to 2014 were reviewed and analyzed along with the information published before. The measurements reviewed include body height, body weight, body mass index and body composition.The severity and duration of bronchial asthma, along with the inhaled corticosteroid treatment, negatively correlate with the height of the children. Also, the fat free mass of the body is decreased and fat mass is increased. Due to more robust body build and the body mass index in children with bronchial asthma is usually increased if compared to healthy children. An increased body mass index in these children reduces the effectiveness of the treatment. Increased body mass index can also be a predictor for the risk for developing late onset bronchial asthma in girls.

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Section: Resultsmentioning

confidence: 99%

Review of different anthropometric and nutritional measurements in children with bronchial asthma

Lipsberga

Kažoka

2015

PoA

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“…A idade da menarca foi de 14 ± 1,4 anos, sendo a menor idade de 9,8 anos e a maior de 17 anos. Portanto, neste grupo de pacientes avaliado, apesar do controle glicêmico inadequado, não foi observado atraso da puberdade, como descrito em outras doenças crônicas, principalmente aquelas com mau controle (34)(35)(36).…”

Section: Discussão E Conclusõesunclassified

Crescimento puberal e altura final em 40 pacientes com diabetes mellitus tipo 1

Meira

Morcillo

Lemos-Marini

et al. 2005

Arq Bras Endocrinol Metab

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RESUMOObjetivo: O diabetes mellitus tipo 1 (DM1) é a mais importante doença endócrino-metabólica crônica entre crianças e adolescentes, podendo ser causa de atraso do crescimento e da puberdade. Avaliamos a influência do DM1 no estirão de crescimento e na puberdade de indivíduos com doença iniciada antes ou no começo desta fase. Casuística e Métodos: Foram avaliados retrospectivamente os prontuários de 40 pacientes (25 meninas), que apresentavam altura final, com coleta dos dados para o cálculo do tempo total de doença, os escores de DP da altura e do peso a cada consulta, a altura-alvo parental, as velocidades de crescimento (VC) anuais, o pico do estirão de crescimento, a duração da puberdade, o ganho total de crescimento durante o estirão puberal e a hemoglobina glicada (HbG). Resultados: Em relação à adequação da altura final para a altura-alvo parental, 37 pacientes ficaram dentro da previsão e 3 ficaram abaixo do limite inferior. Não houve associação entre as variáveis analisadas com exceção da VC no pico do estirão, quando esta foi menor ou igual a 6cm/ano. A idade de início do DM1 e do pico do estirão puberal coincidiram com a faixa etária descrita na literatura; não foi observado atraso na idade de início da puberdade. No entanto, o ganho de altura durante a puberdade foi menor que o descrito na literatura, o mesmo ocorrendo em relação ao pico de VC puberal. De acordo com os valores da HbG, todos os pacientes avaliados apresentaram um controle crônico ruim do DM1. Conclusões: Neste grupo de DM1 com controle inadequado da doença, houve um prejuízo na altura final em relação à altura do início do DM1, provavelmente causado por uma baixa VC durante a puberdade, que, no entanto, não teve influência na altura final em relação ao alvo parental. Objective: Type 1 diabetes mellitus (DM1), the most important chronic endocrine-metabolic disease in children and adolescents, may lead to delayed growth and puberty. In this study we analyzed the influence of DM1 on growth spurt and puberty of patients whose onset of the disease was before or at the beginning of this phase. Patients and Methods: Data from 40 patients, 25 females, who had attained final height were retrospectively obtained, including duration of disease, patient's height and weight SDS at each consultation, parental target height, yearly growth velocities (GV), peak of growth spurt, duration of puberty, magnitude of growth spurt and glycated hemoglobin (HbA 1C ) levels. Results: 37 patients had an adequate final height to parental target height, and only 3 were below the lower limit. There was no significant association among the variables and the appropriate final height to the target height, except for GV on growth spurt, when it was lower than or equal to 6cm/year. The age of onset of DM1 and the age of peak of growth

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“…In cases of severe asthma, the chronic administration of corticoids is responsible for the alterations in growth and the pubertal delay [8]. In coeliac disease [9], cystic fibrosis and chronic inflammatory intestinal disease [10], the fundamental pathological mechanism is the malnutrition. Chronic renal insufficiency and nephrotic syndrome have variable pathogenic mechanisms [11].…”

Section: Pubertal Delaymentioning

confidence: 99%

Diagnosis of Late Puberty

Argente¹

1999

Horm Res Paediatr

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Late puberty is defined as the lack of pubertal development at two standard deviations above the mean age for the general population of the geographical area. In practical terms, this is a chronological age of 14 years for males (testicular volume <4 ml) and 13 years for girls (lack of thelarche). The goal of the assessment is to determine whether the delay or lack of development is due to a lag in normal pubertal maturation or represents an abnormality that must be investigated. Etiologies of pubertal delay and pubertal failure include: a) Constitutional delay of puberty (healthy patients with a clinical history of delayed growth and development; b) Hypogonadotropic states (congenital abnormalities, tumours, endocrinopathies); c) Hypergonadotropic states (chromosomal alterations, syndromes, genetic disorders, radiotherapy/chemotherapy); d) Secondary to chronic illness (organic abnormalities, oncological diseases, malnutrition, eating disorders and endocrinopathies). Diagnostic evaluation must include: a detailed physical examination, including auxological parameters (height and bone maturation), personal and familial antecedents, measurements of general hematological and biochemical parameters, gonadotropins, prolactin, thyroid hormones, sex steroids, growth hormone and growth factors. When necessary, an MRI must be performed. A karyotype is indicated in girls with delayed puberty and short stature and in boys who have small testes and hypergonadotropism.

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2 The growth of children with chronic paediatric disease

Cited by 41 publications

References 92 publications

Review of different anthropometric and nutritional measurements in children with bronchial asthma

Review of different anthropometric and nutritional measurements in children with bronchial asthma

Crescimento puberal e altura final em 40 pacientes com diabetes mellitus tipo 1

Diagnosis of Late Puberty

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