A Case of Painless Thyroiditis Associated With Transient Hyperthyroidism and Periodic Paralysis

Uehara, Toru; Odajima, Hiroshi; Kurosawa, Motohiro; Kunugi, Shyori; Nishioka, Toshio; Sugiyama, Tadashi; Ike, Aiko; Fukuda, Reiko; Shimomura, Yoshiharu; Yamada, Mamoru; Kobayashi, Setsuo

doi:10.2169/naika.66.1747

Cited by 3 publications

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“…Most reported cases are associated with Graves' disease (5). Only a few cases have been described in patients with hyperthyroidism due to other diseases (6)(7)(8). It is thought that pituitary tumors rarely secrete TSH, but this phenomenonhas been increasingly recognized as a cause of hyperthyroidism (9).…”

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A case of thyrotropin (TSH)-secreting tumor complicated by periodic paralysis.

et al. 1990

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A 27-year-old man had symptoms of hyperthyroidism and periodic paralysis. While hyperthyroid, his serum thyrotropin (TSH) level was inappropriately elevated at 6.4/iU/ml. The serum alpha subunit level was also elevated. MRimaging revealed a pituitary tumor and transsphenoidal adenomectomywas performed. Immunocytochemistry with an antibody directed against the beta-subunit of TSHrevealed a TSH-secreting tumor.This is the first case of hyperthyroidism due to a TSH-secreting pituitary tumor complicated by periodic paralysis. This association indicates that thyrotoxicosis may induce paralysis in susceptible persons by a mechanismwhich is not autoimmune.

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A case of thyrotropin (TSH)-secreting tumor complicated by periodic paralysis.

et al. 1990

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“…The most common cause of TPP in thyrotoxicosis is the Graves' disease (1). Only one patient with TPP due to a painless thyroiditis (PT) has been reported (2). Here we describe an additional patient with TTP due to PT who had polymorphic ventricular tachycardia.…”

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confidence: 87%

Thyrotoxic Periodic Paralysis Presenting as Polymorphic Ventricular Tachycardia Induced by Painless Thyroiditis

Lee¹,

Sohn²,

Son³

et al. 2009

Thyroid

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Silent thyroiditis with thyroid-stimulation-blocking antibodies(TSBAb).

et al. 1990

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A 24-year-old man showed thyrotoxic symptoms with hypokalemic periodic paralysis. Serum thyroid hormone levels were high and thyrotropin (TSH) was undetectable. 123I-thyroidal uptake was suppressed. TSH-binding inhibitor immunoglobulin (TBII) was positive. After a month without any treatment, he became hypothyroid. Thyroid hormone level was decreased and TSHwas increased to above the normal range. 123I-thyroidal uptake was increased. TBII activity was still positive. From the clinical findings, a diagnosis of silent thyroiditis was made. Sera obtained in the hypothyroid state revealed the presence of thyroid-stimulationblocking antibodies (TSBAb), but there were no thyroid-stimulating antibodies (TSAb). These results suggest that the hypothyroidism in this patient was due to the presence of TSBAbwith TBII activity.

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A Case of Painless Thyroiditis Associated With Transient Hyperthyroidism and Periodic Paralysis

Cited by 3 publications

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A case of thyrotropin (TSH)-secreting tumor complicated by periodic paralysis.

A case of thyrotropin (TSH)-secreting tumor complicated by periodic paralysis.

Thyrotoxic Periodic Paralysis Presenting as Polymorphic Ventricular Tachycardia Induced by Painless Thyroiditis

Silent thyroiditis with thyroid-stimulation-blocking antibodies(TSBAb).

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