A case of progressive macular hypomelanosis treated with narrow‐band UVB

Yl, Chung; B, Goo; Chung, Wookyung; Gs, Lee; Sk, Hann

doi:10.1111/j.1468-3083.2006.02087.x

Cited by 16 publications

(1 citation statement)

References 3 publications

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“…A recent study of 45 patients with intraperson comparison of two treatment strategies—5% benzoyl peroxide hydrogel/1% clindamycin lotion in combination with UVA irradiation versus 0.05% fluticasone propionate cream in combination with UVA irradiation—found the antibacterial treatment superior (photometric measurements P = .007, patient assessment P < .0001, and dermatologist assessment P < .0001) [7]. PUVA und UVB therapy was reported to achieve improvement [8], at least transiently in a few cases. Anyhow, sometimes the PMH regresses spontaneously within a few years.…”

Section: Discussionmentioning

confidence: 99%

Progressive Macular Hypomelanosis: A Rarely Diagnosed Hypopigmentation in Caucasians

Neynaber

Kirschner

Kamann

et al. 2009

Dermatology Research and Practice

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A 35-year-old woman who developed whitish macules on trunk and limbs at 12 years of age and observed a remarkable increase of the hypopigmentated lesions after her pregnancies at ages 29 and 32 years. Because of the highly characteristic clinical aspect and the light- and electron-microscopic histopathologic findings, we diagnosed progressive macular hypomelanosis (PMH). It is a nonscaly disorder with hypopigmented macules mainly on the trunk and is more often seen in young women. In contrast to some authors assuming the presence of Propionibacterium spp. as a matter of principle in PMH, we report a case with no evidence for Propionibacterium spp.

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Section: Discussionmentioning

confidence: 99%

Progressive Macular Hypomelanosis: A Rarely Diagnosed Hypopigmentation in Caucasians

Neynaber

Kirschner

Kamann

et al. 2009

Dermatology Research and Practice

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Acquired Pigmentary Disorders

Geel

Speeckaert

2016

Rook's Textbook of Dermatology, Ninth Edition

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Abnormal skin pigmentation represents a common skin problem which may be congenitally determined or acquired during life. The former are discussed in Chapter [70]. Acquired pigmentary disorders encompass a broad range of diseases resulting not only in abnormal melanin pigmentation of the skin but also in skin colour change produced by a variety of other endogenous and exogenous pigments. The first part of this chapter examines the melanocyte and normal melanogenesis. The second part discusses not only acquired abnormalities of skin pigmentation resulting from disturbances in melanin pigmentation but also those disorders due to other endogenous or exogenous pigments. The third part describes acquired hypomelanosis including disorders with reduced pigmentation (hypopigmentation) and total absence of pigmentation (depigmentation). In the final part, non‐melanin pigmentation of the skin is examined.

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Acquired Pigmentary Disorders

van Geel,

Speeckaert

2024

Rook's Textbook of Dermatology

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Skin pigmentation by melanocytes is regulated by complex mechanisms including melanoblast migration, melanosome transport, melanosome transfer and melanogenesis. Melanogenesis is influenced by UV light, and endocrine, paracrine and autocrine factors. This leads to a large differential diagnosis of pigmentary disorders. Additionally, melanocytes are highly immunogenic and can be targeted by the immune system, leading to vitiligo and halo naevi. Non‐melanin pigmentation arises from endogenous sources such as haemosiderosis, carotenoderma and ochronosis or exogenous pigmentation such as metals, mediation and tattoos.

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A case of progressive macular hypomelanosis treated with narrow‐band UVB

Cited by 16 publications

References 3 publications

Progressive Macular Hypomelanosis: A Rarely Diagnosed Hypopigmentation in Caucasians

Progressive Macular Hypomelanosis: A Rarely Diagnosed Hypopigmentation in Caucasians

Acquired Pigmentary Disorders

Acquired Pigmentary Disorders

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