1956
DOI: 10.1136/jnnp.19.1.21
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A Clinical Correlation Between Encephalopathy and Papilloedema in Addison's Disease

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1957
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Cited by 60 publications
(15 citation statements)
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“…Case I) and in another an equally serious illness occurs when the blood chemistry is far less disordered (cf. Case 2 and Case 1 in Jefferson, 1956). In Case 1 of this paper there was no complicating factor whilst in the other two cases air had been injected inside the skull, and it is possible that hypothalamic disturbances contributed to the onset of the crisis.…”
Section: Discussionmentioning
confidence: 97%
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“…Case I) and in another an equally serious illness occurs when the blood chemistry is far less disordered (cf. Case 2 and Case 1 in Jefferson, 1956). In Case 1 of this paper there was no complicating factor whilst in the other two cases air had been injected inside the skull, and it is possible that hypothalamic disturbances contributed to the onset of the crisis.…”
Section: Discussionmentioning
confidence: 97%
“…Rarely, in the presence of adrenal cortical hypofunction headache may result from the effects of cerebral oedema associated with adrenal failure. This subject has recently been discussed and illustrated elsewhere (Jefferson, 1956).…”
Section: Discussionmentioning
confidence: 98%
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“…DISCUSSION There is evidence that, in man, alterations in cerebral water content may be associated with disease of the adrenal glands. Jefferson (1956), for example, has described gross cerebral oedema in a patient dying from Addison's disease, and Walsh (1952) observed that papilloedema may occur in this condition. It is of considerable interest in this respect that Benson & Pharoah (1960) have reported benign cerebral hypertension in children who have been given adrenal steroids, and they suggest this state may arise as a steroid withdrawal syndrome.…”
Section: Resultsmentioning
confidence: 97%
“…Pseudotumor cerebri is diagnosed by the modified Dandy's criteria, which include: (1) signs and symptoms of increased intracranial pressure, (2) lack of focal neurologic signs, (3) negative neuroimaging studies, (4) documented increased intracranial pressure, and (5) lack of primary structural or systemic causes of elevated intracranial venous sinus pressure. 2 The exact incidence of Addison's disease, or primary adrenal insufficiency, in children is not known.…”
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confidence: 99%