A fatality caused by accidental production of hydrogen sulfide

Chaturvedi, Arvind; Smith, Dudley R.; Canfield, Dennis V.

doi:10.1016/s0379-0738(01)00552-7

Cited by 25 publications

(14 citation statements)

References 7 publications

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“…In previous reports [5][6][7]9,10,13], the blood concentration of thiosulfate was higher than that of sulfide in 11 of the 12 cases examined. In this study, however, blood concentrations of thiosulfate and sulfide were compared in 14 cases, and the latter was higher than the former in 8 cases (Cases 2-4, 6,8,10,13,14). The mean sulfide concentration in blood of the eight cases was 0.344 AE 0.377 mmol/ml (mean AE SD), which was significantly higher than that of the remaining six cases, 0.019 AE 0.025 mmol/ml (p < 0.05, t-test).…”

Section: Discussionmentioning

confidence: 80%

“…Although many cases of fatal hydrogen sulfide poisoning have been reported , in most of these cases, it resulted from accidents in sewer systems, excrement disposal institutions, chemical factories, volcanoes and hot-spring areas [1][2][3][4][5][6][7][8][9][10][11][15][16][17][18][19][20][21]. To our knowledge, there are few reports of fatal poisoning due to the inhalation of hydrogen sulfide gas that was intentionally generated [13,14].…”

Section: Introductionmentioning

confidence: 99%

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Toxicological analysis of 17 autopsy cases of hydrogen sulfide poisoning resulting from the inhalation of intentionally generated hydrogen sulfide gas

Maebashi

Iwadate

Sakai

et al. 2011

Forensic Science International

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Section: Discussionmentioning

confidence: 80%

Section: Introductionmentioning

confidence: 99%

Toxicological analysis of 17 autopsy cases of hydrogen sulfide poisoning resulting from the inhalation of intentionally generated hydrogen sulfide gas

Maebashi

Iwadate

Sakai

et al. 2011

Forensic Science International

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“…When it comes in contact with an acidic material, H 2 S gas is produced Forensic Science International 187 (2009) e19-e21 [1,[10][11][12]. In this case, it is possible that after the esterification reaction, the residual H 2 SO 4 might have reacted with H 2 S. Therefore, it is highly probable that both NaSH and H 2 SO 4 played the main role in H 2 S production.…”

Section: Discussionmentioning

confidence: 99%

Discolouration of the brain as the only remarkable autopsy finding in hydrogen sulphide poisoning

Park¹,

Zhang²,

Hwang³

2009

Forensic Science International

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“…Postmortem levels of blood sulfide detected by ion-specific electrodes in confirmed lethal H 2 S exposures have ranged from 1.7-3.75 µg/mL. Other authors suggest normal blood sulfide concentrations of 0.03 µg/ml and suggested lethal levels, determined from autopsy samples, at 0.8-5.0 µg/ml [48,53]. Limitations of post-mortem sulfide values include age of samples, sample transport mechanism, duration of exposure, in vivo post mortem metabolism of hydrogen sulfide, and putrefaction of the deceased with microbial hydrogen sulfide production [19].…”

Section: What Diagnostic Studies Confirm H 2 S Toxicity?mentioning

confidence: 99%

Case files of the university of cincinnati fellowship in medical toxicology: Two patients with acute lethal occupational exposure to hydrogen sulfide

Policastro

Otten

2007

J. Med. Toxicol.

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We are presenting cases of 2 employees of a chemical plastics manufacturing plant who experienced acute, brief, inhalational exposure to lethal concentrations of hydrogen sulfide in a confined space. Eight other coworkers not present in the immediate exposure area also presented to local health care facilities with mild complaints of headache, nausea, and dizziness, but they suffered no significant sequelae.Patient #1 was a 55-year-old man who, while working in a confined space, uncoupled a nozzle from a steam pipe, collapsed within one minute, and suffered cardiopulmonary arrest. He had a prior history of asthma, hyperlipidemia, coronary artery disease, and was taking lisinopril, aspirin, and lipitor. Following chest compressions and rescue breathing, both by on-scene responders and EMS, the patient's blood pressure was 210/110 mmHg, the pulse was 100 bpm, and ventilation was assisted at 14/minute. On examination, the patient was minimally combative, with a Glasgow coma score of 6 (E1, V1, M4), with pupils 3 mm bilaterally and reactive to light. The patient had a small partial-thickness burn to the right chest, presumed to be a thermal burn from steam or the hot pipe. No cyanosis or pulmonary crackles were noted. Because of respiratory distress, the patient was intubated on-scene using fentanyl, diazepam, and vecuronium by the combined nurse-physician Air EMS flight team. Post-intubation vital signs were as follows: blood pressure 130/69 mmHg, pulse 95 bpm, respirations 14/min (assisted), and oxygen saturation 98% on 100% FiO 2 . The patient's temperature on arrival to the Emergency Department (ED) was 98.2°F (36.8°C), where his examination remained unchanged.Diagnostic studies were obtained in the ED. Arterial blood gas analysis on 100% O 2 showed: pH 7.24, pCO 2 58 mmHg, pO 2 284 mmHg, oxyhemoglobin 97.2%, deoxyhemoglobin 2.0%, methemoglobin 0.3%, and carboxyhemoglobin 0.0%. The blood appeared red. An electrocardiogram revealed a narrow complex tachycardia, without Q waves, ST-segment abnormalities, or interval changes. Serum chemistries were as follows: a sodium of 139 mEq/L, potassium of 4.2 mEq/L, chloride of 105 mEq/L, bicarbonate of 25 mEq/L, BUN of 15 mg/dL, creatinine of 0.9 mg/dL, and glucose of 156 mg/dL. Creatinine kinase (CK) was 299 IU/L, CK-MB 2.2 IU/L, and troponin-I was 0.05 IU/L. A complete blood count showed: white blood cells at 9.0 k/mm 3 , hemoglobin at 13.0 g/dL, hematocrit at 38.2%, and platelets at 130/mm 3 . Coagulation studies were normal, and a chest radiograph showed adequate endotracheal tube placement, absence of pulmonary edema, and a widened mediastinal silhouette, likely due to projection artifact on an antero-posterior film.Patient #2 was a 53-year-old male co-worker, with a history of hypertension, but he was not receiving medical therapy. He was

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A fatality caused by accidental production of hydrogen sulfide

Cited by 25 publications

References 7 publications

Toxicological analysis of 17 autopsy cases of hydrogen sulfide poisoning resulting from the inhalation of intentionally generated hydrogen sulfide gas

Toxicological analysis of 17 autopsy cases of hydrogen sulfide poisoning resulting from the inhalation of intentionally generated hydrogen sulfide gas

Discolouration of the brain as the only remarkable autopsy finding in hydrogen sulphide poisoning

Case files of the university of cincinnati fellowship in medical toxicology: Two patients with acute lethal occupational exposure to hydrogen sulfide

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