A Preliminary Report on the Injurious Effect of Sodium Bicarbonate in Chicks

Witter, J. F.

doi:10.3382/ps.0150256

Cited by 7 publications

(1 citation statement)

References 2 publications

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“…Scott et al (1944) also found that high dosage of sodium citrate caused not only severe renal damage with tubular dilatation, cast formation and typical "gout rosettes" but also hyperuricaemia and visceral gout. Similar observations were made with sodium bicarbonate by Witter (1936), Bokori (1966a), Mazurkiewicz et al (1970) andChristopher (1977) and with monosodium glutamate by Shimizu et al (1971). The latter workers published colour photographs of the macroscopic renal and visceral lesions and they also showed that the monosodium glutamate effect could be countered with monopotassium glutamate.…”

Section: Sodium: Potassium Imbalacesupporting

confidence: 63%

Renal pathology of the fowl — A review

1981

Avian Pathology

149

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On the basis of published literature and some original observations a comprehensive review is presented of the current concept of renal diseases in the fowl. All aspects of this important but much neglected subject are considered including autolysis, intoxications, inflammatory and degenerative conditions, specific viral diseases, gout, congenital abnormalities and neoplasms. The aetiology and pathogenesis of some forms of avian kidney disease are well understood but there are large gaps in our knowledge on others.

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Section: Sodium: Potassium Imbalacesupporting

confidence: 63%

Renal pathology of the fowl — A review

1981

Avian Pathology

149

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Toxopathology of gout induced in laying pullets by sodium bicarbonate toxicity

Mubarak

Sharkawy

1999

Environmental Toxicology and Pharmacology

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Excess sodium bicarbonate in the diet and its effect on leghorn chickens

Davison

Wideman

1992

British Poultry Science

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1. A commercial 62-week-old layer flock experienced an acute drop in egg production and an increase in shell-less egg production within 2 days of consuming feed erroneously formulated to contain over 30 g/kg instead of 3 g/kg sodium bicarbonate (NaHCO3). Other symptoms included increased water consumption, diarrhoea and increased mortality associated with visceral gout. 2. An experiment was conducted to assess the responses of hens under controlled conditions. Twenty Dekalb XL Single Comb White Leghorn hens (50 weeks old) were placed in individual cages, having ad libitum access to water from trough waterers. Ten hens were fed the TEST (High NaHCO3) feed for one week (Test group), and ten hens remained on normal commercial layer ration (Control group). 3. Hens in the Test group had high water consumption and watery droppings, but egg production and mortality were not affected. Physiological evaluations indicated the Test feed caused metabolic alkalosis. Plasma sodium, urine pH and urinary sodium excretion were increased, and glomerular filtration rates were decreased in the Test group. 4. These physiological effects are consistent with known responses to excess sodium intake in domestic fowl. The reduced egg production and increased mortality caused by the Test feed under commercial conditions may be related to more severe dehydration experienced by hens in multi-bird cages supplied by cup-type watering systems.

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A Preliminary Report on the Injurious Effect of Sodium Bicarbonate in Chicks

Cited by 7 publications

References 2 publications

Renal pathology of the fowl — A review

Renal pathology of the fowl — A review

Toxopathology of gout induced in laying pullets by sodium bicarbonate toxicity

Excess sodium bicarbonate in the diet and its effect on leghorn chickens

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