A prevention programme for somatoform disorders is effective for affective disorders

Garcı́a-Campayo, Javier; Arévalo, E.; Claraco, Luis Manuel; Alda, Marta; Hoyo, Yolanda López del

doi:10.1016/j.jad.2009.06.031

Cited by 9 publications

(6 citation statements)

References 35 publications

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“…We also have noted that the most prevalent psychiatric disorders in primary care are affective (35.8%), anxiety (25.6%), and somatoform (28.8%) disorders, and that 11.5% of patients in this setting present with comorbidity among affective, anxiety, and somatoform disorders [35]. In addition, we have even confirmed that intervention programs for the prevention of somatoform disorders decrease the prevalence of anxiety and depressive disorders at 5-year follow-up [36]. The conclusion is that the overlap among anxiety, depression, and somatic symptoms is so prevalent and consistent that it should be taken into account in psychiatric classifications [35].…”

Section: Etiologic Explanation For the Joint Hypermobility Syndrome-asupporting

confidence: 71%

Joint Hypermobility and Anxiety: The State of the Art

Campayo

Asso

Alda

2010

Curr Psychiatry Rep

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Joint hypermobility (JH) is considered a common benign, hereditary, overlap, connective tissue disorder with a prevalence in the general population of about 10% in European populations and 25% in other ethnic groups. JH shows an association with mitral valve prolapse and fibromyalgia. However, the most significant and important association between joint hypermobility syndrome (JHS) and any other disorder from a clinical point of view is with panic disorder. This article summarizes all published studies on JHS and anxiety, analyzing the main results and limitations. An overview of the etiologic explanation of the association between JH and anxiety, with special focus on genetic findings, is also included. The most relevant conclusions are the following: JHS is more prevalent in individuals with panic disorder/agoraphobia, and patients with JHS present with greater prevalence of panic disorder/agoraphobia. In addition, there is an association between JHS severity and severity of anxiety, and mitral valve prolapse plays a secondary role in the association between JHS and anxiety. New fields of research based on these data are suggested.

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Section: Etiologic Explanation For the Joint Hypermobility Syndrome-asupporting

confidence: 71%

Joint Hypermobility and Anxiety: The State of the Art

Campayo

Asso

Alda

2010

Curr Psychiatry Rep

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“…We only included 1 RCT 40 of psychological and educational interventions in adolescents to prevent depression in primary care; when this setting restriction was not used, however, most trials were limited to children and adolescents. 23 In only 4 RCTs [43][44][45]47 were intervention clinicians primary care staff, and their pooled effect size (SMD = -0.197) was not different when compared with mental health providers (SMD = -0.141); although there were too few RCTs to draw conclusions. We excluded 1 RCT 51 because the comparator was not care as usual, waiting list, or placebo; in both arms of this RCT, however, primary care physicians implemented interventions to prevent depression (motivational interviewing vs brief advice to engage adolescents with an Internet-based depression program).…”

Section: Discussionmentioning

confidence: 99%

“…The 14 RCTs [36][37][38][39][40][41][42][43][44][45][46][47][48][49][50] were published between 1993 and 2016. Six RCTs were conducted in the United States, 3 in Spain, 2 in the Netherlands, 2 in the United Kingdom and 1 in China; 9 were aimed at adults and 5 at specific populations.…”

Section: Characteristics Of Included Studiesmentioning

confidence: 99%

“…The pooled SMD using the first evaluation over time (posttreatment) of each RCT was similar to the last evaluation (Table 3). When we excluded from the analysis the only RCT that used the reduction of depressive symptoms as a secondary outcome, 43 the pilot study of Brugha, 45 or both studies, 43,45 the pooled SMDs were then similar (Table 3).…”

Section: Sensitivity Analysesmentioning

confidence: 99%

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Effectiveness of Psychological and Educational Interventions to Prevent Depression in Primary Care: A Systematic Review and Meta-Analysis

et al. 2017

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PURPOSE Although evidence exists for the efficacy of psychosocial interventions to prevent the onset of depression, little is known about its prevention in primary care. We aimed to evaluate the effectiveness of psychological and educational interventions to prevent depression in primary care. METHODSWe conducted a systematic review and meta-analysis of relevant randomized controlled trials (RCTs) examining the effect of psychological and educational interventions to prevent depression in nondepressed primary care attendees. We searched MEDLINE, PsycINFO, Web of Science, OpenGrey Repository, Cochrane Central Register of Controlled Trials, and other sources up to May 2016. At least 2 reviewers independently evaluated the eligibility criteria, extracted data, and assessed the risk of bias. We calculated standardized mean differences (SMD) using random-effects models. RESULTSWe selected 14 studies (7,365 patients) that met the inclusion criteria, 13 of which were valid to perform a meta-analysis. Most of the interventions had a cognitive-behavioral orientation, and in only 4 RCTs were the intervention clinicians primary care staff. The pooled SMD was -0.163 (95%CI, -0.256 to -0.070; P = .001). The risk of bias and the heterogeneity (I 2 = 20.6%) were low, and there was no evidence of publication bias. Meta-regression detected no association between SMD and follow-up times or SMD and risk of bias. Subgroup analysis suggested greater effectiveness when the RCTs used care as usual as the comparator compared with those using placebo.CONCLUSIONS Psychological and educational interventions to prevent depression had a modest though statistically significant preventive effect in primary care. Further RCTs using placebo or active comparators are needed. Ann Fam Med 2017;15:262-271. https://doi.org/10.1370/afm.2031. INTRODUCTIONA ccording to the World Health Organization, major depressive disorder affects about 350 million people worldwide. 1 An international study found the average 12-month prevalence of major depression to be 5%. 2 In primary care, though, this figure is higher (11%).3 About 60% to 70% of patients with acute depression experience suicidal ideas, with the actual incidence of suicide in depressive patients being 10% to 15%. 4 Patients with a specific physical illness who develop depression have an excess mortality of 52%. 5 In terms of global disease burden measured in disability-adjusted life years, major depressive disorder increased by 37% between 1990 and 2010 6 and is projected to become the single leading cause of disease burden by 2030 in high-income countries. 7 Two ways exist to reduce the disease burden of depression: treatment and prevention of new cases. Despite effective treatments for depression, the disease burden of depression can be reduced by only 20%; not all cases are recognized, and when recognized, not all patients receive appropriate therapy or adhere to treatment. 263In recent decades interest in prevention has grown, 9 and it is now a key objective in the strategic plan ...

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“…Nevertheless, there is no consensus on the best therapeutic approach, and treatment of FM is a challenge for clinicians [10]. Instead, an integrated biopsychosocial approach that includes both nonpharmacological and pharmacological therapies improves outcomes [11][12][13][14][15][16][17][18][19][20][21]. Multidisciplinary programs for FM typically include educational, cognitive, and behavioral strategies, physical training, and medication [9,22,23].…”

Section: Introductionmentioning

confidence: 99%