Periodontal disease, which mainly includes gingivitis and periodontitis, is caused by pathological changes in periodontal tissue. It is highly prevalent in adults and is more severe in the elderly group as an estimated 10-15% of this group will develop severe periodontitis (Ebersole et al., 2016). Periodontitis progresses with the accumulation of plaque, dysbiosis of bacterial ecology, formation of periodontal pockets, gum recession, tissue destruction, and loss of alveolar bone, eventually leading to tooth loss (Morelli et al., 2020).There is clear evidence that periodontal disease is associated with inflammatory markers, cardiovascular disease, and digestive system tumours (Carrizales-Sepúlveda et al., 2018;Li et al., 2020;Șurlin et al., 2020). Improving individual modifiable risk factors is a reliable way to prevent periodontal disease, while public health interventions are necessary to be indeed effective and sustainable under real-life conditions (Janakiram & Dye, 2020;Lobbezoo et al., 2020).Short sleep duration has been shown to be a risk factor for a variety of health outcomes (Itani et al., 2017). Meanwhile, short sleep was associated with increased infection risk and could activate inflammatory signalling pathways (Irwin, 2019). The association between short sleep and periodontal disease has been reported in many studies, but