A sudden presentation of abdominal compartment syndrome

Abstract

LeTTeRs TO The eDITOR Dear editor, Abdominal compartment syndrome (ACs) is defined as sustained intraabdominal pressure (IAP) exceeding 20 mm hg, which causes end-organ damage due to impaired tissue perfusion, as with other compartment syndromes [1, 2]. This dysfunction can extend beyond the abdomen to other organs like the heart and lungs. ACs is most commonly caused by trauma or surgery to the abdomen. It is characterised by interstitial oedema, which can be exacerbated by large fluid shifts during massive transfusion of blood products and other fluid resuscitation [3]. Normally, IAP is nearly equal to or slightly above ambient pressure. Intra-abdominal hypertension is typically defined as abdominal pressure greater than or equal to 12 mm hg [4]. Initially, the abdomen is able to distend to accommodate the increase in pressure caused by oedema; however, IAP becomes highly sensitive to any additional volume once maximum distension is reached. This is a function of abdominal compliance, which plays a key role in the development and progression of intraabdominal hypertension [5]. surgical decompression is required in severe cases of organ dysfunction-usually when IAPs are refractory to other treatment options [6]. excessive abdominal pressure leads to systemic pathophysiological consequences that may warrant admission to a critical care unit. These include hypoventilation secondary to restriction of the deflection

Bladder compliance dynamics of pelvic organ prolapse patients subjected to robotic-assisted sacrocolpopexy

“…A non-physiological stretch of part of the detrusor shifts the length-tension curve to a higher tension, thereby results in a higher pressure, in response to lling volume [28]. This proposal is supported by a clinical study showing a sudden onset bladder compression results in an abruptly elevated intra-vesical pressure in patients [29] and a preclinical study demonstrated modi ed bladder geometry that abolishes physiological distension results in decreased bladder compliance in rabbits [30]. Notably, after correcting anatomical abnormalities in POP patients, RSCP relieved the restriction of bladder expansion coming from the prolapsed organ.…”