A test of nerve conduction to muscles of the shoulder girdle as an aid in the diagnosis of proximal neurogenic and muscular disease

Gassel, M. M.

doi:10.1136/jnnp.27.3.200

Cited by 49 publications

(20 citation statements)

References 6 publications

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“…6,7,14,18 The distal latency values found on electrodiagnostic testing read as negative for SSN in this study are similar to those reported in normal participants by Rosenfalck and Rosenfalck, 31 Kraft, 18 Gassel, 14 and Casazza et al 7 (values reported as 2.6 to 2.76 milliseconds for the supraspinatus and 3.3 to 3.41 milliseconds for the infraspinatus in needle studies), but are lower than those found in a recent report by Buschbacher et al 6 (3.2 and 3.6 milliseconds, respectively; Table II). The noted difference may be secondary to technique because the Buschbacher et al study used surface electrodes, whereas our EMG/NCS were recorded with needle electrodes.…”

Section: Discussionmentioning

confidence: 98%

Suprascapular neuropathy in a shoulder referral practice

Boykin

Friedman

Zimmer

et al. 2011

Journal of Shoulder and Elbow Surgery

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Section: Discussionmentioning

confidence: 98%

Suprascapular neuropathy in a shoulder referral practice

Boykin

Friedman

Zimmer

et al. 2011

Journal of Shoulder and Elbow Surgery

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“…The musculocutaneous nerve originates from the lateral cord of the brachial plexus at or about the lower border of the pectoralis minor muscle and its fibres derive chiefly from the fifth and sixth cervical nerve roots (Sunderland, 1968). Conduction along motor fibres from the axilla to the brachial biceps muscle has been described by Redford (1958) and from Erb's point to the brachial biceps by Gassel (1964) and Kraft (1972). Nelson and Currier (1969) stimulated the nerve at two sites in seven normal subjects and determined the motor conduction velocity along the fibres to the brachial biceps in the nerve segment between the anterior cervical triangle and the axilla.…”

mentioning

confidence: 99%

Motor and sensory conduction in the musculocutaneous nerve.

Trojaborg¹

1976

Journal of Neurology, Neurosurgery & Psychiatry

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SYNOPSIS Motor and sensory conductionvelocity in the musculocutaneous nerve were determined in 51 normal subjects. The maximal velocity from the anterior cervical triangle to the axilla was the same in motor and sensory fibres. The musculocutaneous nerve originates from the lateral cord of the brachial plexus at or about the lower border of the pectoralis minor muscle and its fibres derive chiefly from the fifth and sixth cervical nerve roots (Sunderland, 1968). Conduction along motor fibres from the axilla to the brachial biceps muscle has been described by Redford (1958) and from Erb's point to the brachial biceps by Gassel (1964) and Kraft (1972). Nelson and Currier (1969) stimulated the nerve at two sites in seven normal subjects and determined the motor conduction velocity along the fibres to the brachial biceps in the nerve segment between the anterior cervical triangle and the axilla.The study presented in this report deals with conduction velocity along the musculocutaneous nerve in normal subjects in proximal (fossa supraclavicularis-axilla) and distal segments of motor fibres and in proximal (fossa supraclavicularis-axilla) and intermediate (elbowaxilla)

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“…Die Überleitungszeit des N. axillaris und des N. musculocutaneus wurde mit Routinemethoden bestimmt, unter Verwendung konzentrischer Nadelelektroden mit einem Durchmesser von 0,48 mm [12]. Die Überleitungszeit am N. facialis wurde durch die Bestimmung der Latenz des CMAP am M. orbicularis oris mit Oberflächenelektroden mit kleinen Ableiteflächen ermittelt [13].…”

Section: Elektrophysiologieunclassified

Erbliche motorische und sensorische Neuropathie - Typ LOM (HMSN-L) - Elektrophysiologische Charakteristika

Christova¹,

Alexandrov²,

Krampfl³

et al. 2005

Klin Neurophysiol

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ZusammenfassungUntersuchungen zu den elektrophysiologischen Charakteristika bei einer gröûeren Familie mit erblicher motorischer und sensorischer Neuropathie ± Typ LOM (HMSN-L) mit 27 betroffenen Mitgliedern wurden durchgeführt. Typischerweise tritt bei dieser Erkrankung klinisch eine Gehbehinderung infolge einer fortschreitenden Schwäche der unteren Extremitäten während der frühen Kindheit auf. Später entwickelt sich auch eine Schwäche im Bereich der oberen Extremitäten. Bei annähernd 30 % der Patienten entwickeln sich im Verlauf der zweiten oder dritten Lebensdekade Zeichen einer bulbären Beteiligung sowie eine erhebliche Hörminderung bis hin zur Ertaubung. Die motorische Nervenleitgeschwindigkeit war bei den untersuchten Patienten erheblich verlangsamt, insbesondere am N. musculocutaneus, N. axillaris, N. hypoglossus und N. fazialis. Sensorische Nervenaktionspotenziale waren häufig nicht messbar. Im Verlauf der Kindheit zeigte sich in Verlaufsuntersuchungen eine zunehmende Progression der Leitungsverzögerungen. Bei allen betroffenen Patienten waren regelmäûig multiple A-Wellen nachweisbar. Der Blinkreflex zeigte charakteristische pathologische Antwortpotenziale mit drei Komponenten und verlängerter Latenz. Die HMSN-L kann als demyelinisierende Neuropathie mit schwerem und frühem axonalem Schaden charakterisiert werden. Während der Kindheit zeigte sich ein Fortschreiten der ebenfalls verlangsamten Nervenleitgeschwindigkeiten im Unterschied zum mehr statischen Bild bei der hereditären motorischen und sensorischen Neuropathie Typ I (HMSN-I). AbstractWe measured electrophysiological characteristics in a large family, 27 members of which suffered from hereditary motor and sensory neuropathy of the LOM type (HMSN-L). Typically, in this disease there is in early childhood a clinical aspect of limited walking ability due to progessive weakening of the legs. Later there is also weakness in the arms. In about 30 % of the patients signs of bulbar involvement occurred during the third decade of life, as well as considerable hardness of hearing leading to total deafness. Motor nerve conduction speed was considerably reduced in the investigated patients and facial nerves. Often it was no longer possible to measure sensory nerve action potentials. In the course of childhood nerve conduction velocity declined progressively. Multiple A waves were regularly seen in all the involved response potentials with three components and prolonged latency. HMSN-L can be characterised as a demyelinising neuropathy associated with early and severe axonal damage. During childhood there was a progressive decrease in the already slowed-down nerve conduction velocity in contrast with the more static pattern of Type I of hereditary sensory neuropathy (HMSN-I).

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A test of nerve conduction to muscles of the shoulder girdle as an aid in the diagnosis of proximal neurogenic and muscular disease

Cited by 49 publications

References 6 publications

Suprascapular neuropathy in a shoulder referral practice

Suprascapular neuropathy in a shoulder referral practice

Motor and sensory conduction in the musculocutaneous nerve.

Erbliche motorische und sensorische Neuropathie - Typ LOM (HMSN-L) - Elektrophysiologische Charakteristika

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