A variant of Capgras syndrome with delusional conviction of inanimate doubles in a patient with grandmal epilepsy

Nejad, Alireza Ghaffari; Toofani, Khatereh

doi:10.1111/j.0924-2708.2006.00114.x

Cited by 8 publications

(6 citation statements)

References 14 publications

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“…The striking feature is that he has adjusted well to his family till the age of 21 years, providing further evidence that a mild form of the disease does exist. The presenting complaint of increased aggression and suspicion in this patient could, however, be linked to right hemispheric dysfunction caused by epilepsy, hypothesized to cause paranoid psychoses and Capgras syndrome (13).…”

Section: Discussionmentioning

confidence: 87%

Typical Aicardi syndrome in a male

Saddichha

Manjunatha

Akhtar

2007

Acta Neuropsychiatr.

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Section: Discussionmentioning

confidence: 87%

Typical Aicardi syndrome in a male

Saddichha

Manjunatha

Akhtar

2007

Acta Neuropsychiatr.

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“…The original Ellis and Young hypothesis included all sense modalities , but the fact they specifically used the visual processing of the face to argue their case appears to have led to a split in the literature. Some researchers predominately define the Capgras delusion as being specific to people (Bourget & Whitehurst: 2004;Brighetti et al 2007;Coltheart: 2007;Dietl, Brunner & Friess: 2003;Doran: 1990;Josephs: 2007;Tamam et al: 2003;Young, G. : 2008), whilst others extend the syndrome to include inanimate objects such as household furniture (Ellis et al: 1996;Nejad & Toofani: 2006) and animals, particularly pets ( Ellis et al: 1996;Rosler et al: 2001).…”

Section: Capgras For Inanimate Objectsmentioning

confidence: 99%

The Capgras delusion: an integrated approach

Wise

2015

Phenom Cogn Sci

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Prof. Peter Menzies: principal supervisor from the second half of the first year until thesis submission. I would like to thank Prof. Peter Menzies for his excellent supervision, for asking me the hard questions, and for persuading me to pursue challenges I would have otherwise avoided. I would also like to thank him for his personal support during my treatment for cancer.Dr. Mitch Parsell: principal supervisor for the first six months, and associate supervisor for the remainder of my candidature. I would like to thank Dr. Mitch Parsell for being there for my entire candidature, for always being supportive, encouraging and generous with his time. I would also like to thank him for being a friend when times were challenging.Dr. Robert Sinnerbrink: associate supervisor for the first six months of my candidature. I would like to thank Dr. Robert Sinnerbrink for his support, and for generously reading relevant section of the original German edition of Jaspers' General Psychopathology, and for helping me clarify Jaspers' concepts.Prof. Jeanette Kennett: associate supervisor from the second year of my candidature to submission. I would like to thank Prof. Jeanette Kennett for her advice, her friendly support, and for her reassurance in my moments of doubt.I would also like to express my appreciation to the following academics for their generosity in spending time advising me in an un-official capacity.Dr. Lisa Bortolotti: I would like to thank Dr. Lisa Bortolotti for regularly meeting with me and my principal supervisor during her three months sabbatical at Macquarie University, during the first year of my candidature. Her advice in the early stages of my candidature was extremely valuable.x Assoc. Prof. Robyn Langdon: I would like to thank Assoc. Prof. Robyn Langdon for meeting with me and with my supervisors, and for offering invaluable advice on the cognitive science aspects of my thesis. I would also like to thank her for reading and commenting on several drafts of Chapter 6. She has continued to be encouraging and supportive, and always generous with her time. Prof. Max Coltheart: I would like to thank Prof. Max Coltheart for inviting me to join the Australian Research Council of Excellence in Cognition and its Disorders (CCD) Belief Group at Macquarie University, and for his patience in answering my many questions. I would also like to thank all members of the CCD Belief Group for their regular stimulating and informative discussions, without which this thesis could not have been written in its present form. 1 Revision Washington, DC: American Psychiatric Association, 2000, pxxx "It is something of as fantasy, to use Dennett's term, to suggest that neuroscience or psychology are best done by averaging out, reducing, and re-engineering first-person data so that it looks like third-person data."

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“…These syndromes include Capgras syndrome, Fregoli syndrome, syndrome of intermetamorphosis, syndrome of subjective doubles [54] and syndrome of inanimate doubles [55] . These syndromes are important insofar as they have been shown to have a significant organic contribution [53,56] .…”

Section: Delusional Misidentification Syndromesmentioning

confidence: 99%

The Neurology of Psychosis

Oyebode¹

2008

Med Princ Pract

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The neural basis of psychosis is yet to be fully elucidated. In this review the contribution of schizophrenia-like psychosis of epilepsy, delusional misidentification syndromes and psychotic phenomena, such as auditory and visual hallucinations, to our understanding of the neural basis of psychosis is examined. Schizophrenia-like psychosis of epilepsy is associated with seizures originating from the limbic structures. Reduced seizure frequency, left-sided electrical foci, and neurodevelopmental lesions manifesting as cortical dysgenesis are known to influence the likelihood of developing schizophrenia-like psychosis of epilepsy. The delusional misidentification syndromes are a group of rare psychiatric symptoms in which impairments of face recognition memory are present. These conditions appear also to be associated with organic lesions affecting limbic structures and also involving both the frontal and parietal lobes. There is evidence that right-sided lesions predominate in the aetiology of delusional misidentification syndromes. Thus, the common link between schizophrenia, schizophrenia-like psychosis of epilepsy and delusional misidentification syndromes appears to be involvement of limbic structures in their pathophysiology. Discrete psychotic phenomena such as visual and auditory hallucinations appear to arise from functional changes in the same cortical areas subserving the normal physiological functions of vision and audition but also involving limbic structures. In conclusion, the limbic structures appear to be central to the psychopathology of psychosis but with involvement of frontal and parietal structures. These inquiries are revealing as much about psychosis as they are about the nature of normal brain function.

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A variant of Capgras syndrome with delusional conviction of inanimate doubles in a patient with grandmal epilepsy

Abstract: Previous reports showed Capgras phenomenon as a clinical presentation in epileptic patients. We show that the delusion of doubles of inanimate objects as a variant of Capgras syndrome could also be a clinical presentation in epileptic patients.

Cited by 8 publications

References 14 publications

Typical Aicardi syndrome in a male

Typical Aicardi syndrome in a male

The Capgras delusion: an integrated approach

The Neurology of Psychosis

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