Ablation of Post-Myocardial Infarction Focal Ventricular Tachycardia

Abstract

In the chronic phase of myocardial infarction, the presence of scar areas allows the development of macro-reentries which become the most frequent mechanism underlying ventricular tachycardia (VT). A focal mechanism has been already described in the presence of scar in animal models or in humans but only during surgery. We report a case of focal automatic VT arising from postinfarction scar fibrosis, successfully mapped and ablated during an electro-physiological procedure.