AC magnetization analyses in iron-rich amorphous wires

Abstract

Domain wall depinning and propagation in a long wire of amorphous Fe77.5Si7.5B15 alloy in the as-quenched state are studied during AC magnetization under a DC biasing field produced by a solenoid perpendicular to the wire. The active walls undergoing the depinning and propagation are classified into positive and negative ones. Consequently there are alternating and unidirectional magnetic reversal modes, both having two distinct types. The alternating mode can easily be induced by an external magnetic pole equidistant from the wire ends. The experimental data obtained in this work and published elsewhere are discussed in detail. It is concluded that the eventual depinning of the closure domain wall from the end is induced by its elongation and transverse movements. The latter process is essentially the same as domain wall propagation. Both are dynamic with a dominant eddy current damping and domain wall shape-dependent DC critical field.

A soft magnetic wire for sensor applications

“…During propagation the shape of the wall is quasi-planar [7] or conical [10] according to different authors. Further details on the propagating wall shape, which is several centimetres long in amorphous wires, and on its dynamical aspects can be found elsewhere [26,27]. Figure 5 shows schematically the domain structure during the magnetization reversal process inside the inner core.…”

Influence of tensile stress and frequency on the longitudinal magnetic hysteresis of amorphous wires

“…Magnetization reversal process has been deeply studied and it is assumed to start from the depinning of the wall between closure domains and the main domain of positive magnetization [6]. Thus, when the energy stored at the latter is greater than a critical energy barrier at a given applied field the wall propagates along the wire until reaching the closure structure at the opposite end.…”

Характеристика Гонадотропної Функції У Хворих З Пубертатною Матковою Кровотечею У Сучасному Соціумі

“…Обробка культури нейронів гіпоталамуса інсуліном призводить до значного посилення секреції Гн-РГ цими клітинами [4]. Встановлено, що інсулінова недостатність може впливати на чутливість гонадотропів гіпофіза до дії Гн-РГ і призводити до зниження секреції гонадотропінів [5,6]. Однак є дані про те, що не тільки нестача, але й надлишок інсуліну може зумовити порушення функціонування гіпоталамічних нейронів, наслідком яких є зниження рівня гонадотропінів [7].…”