Pediatric Neurotoxicology 2016
DOI: 10.1007/978-3-319-32358-9_9
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Academic and Psychosocial Impact of Air Pollution on Children

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Cited by 2 publications
(3 citation statements)
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“…An increasing body of evidence is showing associations between traffic-related exposure and diagnoses of psychiatric disorders in children and adolescents ( 17 ), self-regulation, and ADHD ( 18 , 19 ). Although most studies included large, representative samples with clinically relevant community-level results, findings were correlational and most did not control for multiple toxin exposure prenatally or postnatally and moderating factors such as maternal use of psychotropic medications, making it difficult to exclude genetic predispositions.…”
Section: Effects Of Severe Air Pollution On Brain-related Functions Imentioning
confidence: 99%
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“…An increasing body of evidence is showing associations between traffic-related exposure and diagnoses of psychiatric disorders in children and adolescents ( 17 ), self-regulation, and ADHD ( 18 , 19 ). Although most studies included large, representative samples with clinically relevant community-level results, findings were correlational and most did not control for multiple toxin exposure prenatally or postnatally and moderating factors such as maternal use of psychotropic medications, making it difficult to exclude genetic predispositions.…”
Section: Effects Of Severe Air Pollution On Brain-related Functions Imentioning
confidence: 99%
“…Limitations in developmental OA-pollution research involve the incompleteness of findings [( 37 ); see Table 1 ], scientific uncertainty regarding risk assessment [( 38 ); see Table 2 ], and inconsistent research on outcomes of exposure [( 18 ); see Table 3 ]. Table 1 summarizes the recommendations of the National Institute of Environmental Health Sciences/National Institute of Health panel ( 37 ) to fill knowledge gaps, with main priorities to understand the OA-pollution characteristics necessary to elicit detrimental pro-inflammatory responses, confirm the role of neuroinflammation in toxicity, identify all critical mechanisms, cell types, direct and indirect pathways, and their complex interactions (see Figure 1 ).…”
Section: Knowledge Limitations and Future Researchmentioning
confidence: 99%
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