Acid corrosive esophagitis: radiographic findings

Muhletaler, CA; Aj, Gerlock; Soto, L de; Sa, Halter

doi:10.2214/ajr.134.6.1137

Cited by 31 publications

(19 citation statements)

References 5 publications

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“…At three to four weeks after injury, a stricture may form (19, 20). Because perforation, necrosis, and subsequent complications such as mediastinitis may occur during the acute or early states, EBD is usually started at least 1 month after corrosive injury (16).…”

Section: Discussionmentioning

confidence: 99%

Balloon Dilatation for Corrosive Esophageal Strictures in Children: Radiologic and Clinical Outcomes

et al. 2010

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ObjectiveWe retrospectively evaluated the effectiveness of the esophageal balloon dilatation (EBD) in children with a corrosive esophageal stricture.Materials and MethodsThe study subjects included 14 patients (M:F = 8:6, age range: 17-85 months) who underwent an EBD due to a corrosive esophageal stricture. The causative agents for the condition were glacial acetic acid (n = 9) and lye (n = 5).ResultsA total of 52 EBD sessions were performed in 14 patients (range 1-8 sessions). During the mean 15-month follow-up period (range 1-79 months), 12 patients (86%) underwent additional EBD due to recurrent esophageal stricture. Dysphagia improved after each EBD session and oral feeding was possible between EBD sessions. Long-term success (defined as dysphagia relief for at least 12 months after the last EBD) was achieved in two patients (14%). Temporary success of EBD (defined as dysphagia relief for at least one month after the EBD session) was achieved in 17 out of 52 sessions (33%). A submucosal tear of the esophagus was observed in two (4%) sessions of EBD.ConclusionOnly a limited number of children with corrosive esophageal strictures were considered cured by EBD. However, the outcome of repeated EBD was sufficient to allow the children to eat per os prior to surgical management.

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Section: Discussionmentioning

confidence: 99%

Balloon Dilatation for Corrosive Esophageal Strictures in Children: Radiologic and Clinical Outcomes

et al. 2010

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“…Those with gastric body stricture developed postprandial vomiting relatively late because the pyloric ring and antrum were relatively preserved. It seems reasonable to assume that this preservation is related to posture, stomach contents [5, 13, 14, 15, 16], and/or gastric body spasm [17, 18]at the time that patients ingested the acid solution. According to our laboratory data, there was no difference in injury severity for gastric body stricture in comparison to the antral analog.…”

Section: Discussionmentioning

confidence: 99%

Early Surgical Correction for Isolated Gastric Stricture following Acid Corrosion Injury

Tseng

et al. 2002

Dig Surg

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Objective: To evaluate the feasibility and long-term results of early gastric surgery for patients with isolated gastric stricture following acid corrosion injury. Materials and Methods: Upper gastrointestinal (UGI) series was routinely performed around the 4th week after acid corrosion injury. Patients with gastric stricture and no risk of delayed esophageal stricture underwent early solitary gastric surgery, which was defined when performance of the procedure took place within 2 months of the injury. Results: From June 1988 to June 2000, 35 of 378 patients with acid corrosion injury developed isolated gastric stricture. Twenty-four (68.6%) lesions were located in the antrum, and 11 (31.4%) in the gastric body. Postprandial vomiting presented earlier for the antral stricture group (17.6 ± 1.1 versus 25.4 ± 3.4 days after injury; p = 0.005). The UGI series was performed from 16 to 41 days after injury (average 25 days). Of the 35 acid corrosion injury patients in this study, 4 were excluded because of late referrals to our institution or the patient’s hesitation which resulted in delayed surgery. The remaining 31 patients underwent gastric surgery 35.7 ± 3.2 days after ingestion (34.6 ± 3.6 and 38.1 ± 3.4 days for cases of antral and gastric body stricture, respectively). Surgical procedures consisted of hemigastrectomy (n = 16), antrectomy (n = 2), gastroenterostomy (n = 2), subtotal gastrectomy (n = 6), and total gastrectomy (n = 5). There were 4 cases of postoperative complications (12.9%) including adhesion ileus (n = 2), wound infection (n = 1), and massive, postoperative UGI bleeding (n = 1). Surgical mortality was zero. All patients tolerated oral intake well after surgery. During the minimum follow-up period of 1 year, 1 patient developed esophagojejunostomy stenosis, which was resolved by dilation, and there was 1 case of dumping syndrome, which was treated by diet control. Conclusion: Early surgery correction is feasible and safe if patients with isolated gastric stricture following acid corrosion injury are carefully selected. All patients in our study recovered early, with a low morbidity rate.

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“…It can also cause duodenal scarring and narrowing [1,3,4]. The major long-term mortality after caustic ingestion is due to cicatrization of the oesophageal strictures [4] and subsequent dysphasia [7][8][9][10][11]. In grade III corrosive injury involving the lower end of oesophagus, the inflammation and subsequent fibrosis may entrap and compress the anterior and posterior vagal nerves, which is seen in close proximity to the lower end of oesophagus.…”

Section: Introductionmentioning

confidence: 99%