Acquired crateriform hyperkeratotic papules of the feet: An unusual variant of focal acral hyperkeratosis

Ballambat, Sathish Pai; Pai, Kanthilatha

doi:10.4103/0378-6323.35749

Cited by 5 publications

(5 citation statements)

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“…Several therapies have been tried, including liquid nitrogen cryotherapy, keratolytics like urea and salicylic acid, topical and systemic retinoids, and topical and systemic steroids, all with limited results. 9 Of note, 1 case reported sustained improvement after use of topical calcipotriol twice daily for 8 weeks, with no recurrence at follow-up in 8 months. 9 As shown in our case, FAH can present with significant hyperpigmentation, which can be treated with topical lightening creams such as hydroquinone.…”

Section: Discussionmentioning

confidence: 92%

“… 9 Of note, 1 case reported sustained improvement after use of topical calcipotriol twice daily for 8 weeks, with no recurrence at follow-up in 8 months. 9 As shown in our case, FAH can present with significant hyperpigmentation, which can be treated with topical lightening creams such as hydroquinone. Unfortunately, our patient was refractory to topical keratolytics.…”

Section: Discussionmentioning

confidence: 92%

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Focal acral hyperkeratosis with response to acitretin

Mansour,

Wenzel,

Lowe

et al. 2024

JAAD Case Reports

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Section: Discussionmentioning

confidence: 92%

Section: Discussionmentioning

confidence: 92%

Focal acral hyperkeratosis with response to acitretin

Mansour,

Wenzel,

Lowe

et al. 2024

JAAD Case Reports

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“…1,2,9 Since Dowd and Blum's initial description, slightly more than a dozen cases of FAH have been reported. 1,5,[10][11][12][13][14][15][16][17][18][19][20] We reviewed thirteen cases in the literature (from 1996 to 2018). The patients were older with a median age of 35 years (range between 9 and 62 years old).…”

Section: Discussionmentioning

confidence: 99%

Marginal acrokeratoderma: a case series

Ibekwe

Ogunbiyi

2020

Int J Res Dermatol

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<p>Marginal acrokeratoderma (MAK) are complex disorders characterized by distribution of cornified papules/plaques along the dorso-plantar and dorso-palmar junction of the feet and hands. They belong to the family of punctate palmoplantar keratoderma. No associated gene defect has been detected. There are two clinically identical types of hereditary/familial MAK; namely acrokeratoelastoidosis (AKE) and focal acral hyperkeratosis (FAH); differentiated by the presence of elastorrehexis in AKE. The aim of this article is to report MPK lesions that are not included in the familiar groupings of punctate and polygonal papules. We described thirteen cases and reviewed the literature. Average age at presentation was 24 years, age ranged from 5 to 79 years. Majority of our cases had lesions on only their feet, that of FAH had lesions on only their hands and AKE had lesions on both hands and feet. Although most lesions reported in literature with FAH and AKE were characterized as 2-5mm flesh-colored to yellowish flat-topped/polygonal, keratotic/punctate papules, coalescing into plaques, we also reported lesions that were keratotic with cracks, desquamating and cobble-stone in appearance. This case series calls for more studies on variations in clinical presentation of MAK lesions and an opportunity to revisit the genetic basis and investigate triggers of this rare disorder.</p>

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“…However, in a prospective, right/left comparative open study including 24 patients, treatment of one lesion with topical calcipotriol was compared to one lesion that was left untreated. An average treatment duration of 3.9 months resulted in repigmentation in only three of the [66,67], epidermolytic ichthyosis [68], epidermolytic palmoplantar keratoderma [69], erosive pustular dermatosis of the frontal scalp [70], eruptive vellus hair cysts [71], erythema annulare centrifugum [72], extramammary Paget disease [73], focal acral hyperkeratosis [74], granular parakeratosis [75][76][77], Grover disease [78], hand-foot syndrome [79], hyperkeratosis lenticularis perstans [80], ichthyosis linearis circumflex of Netherto syndrome [81], inflammatory linear verrucous epidermal nevus [82][83][84][85], Kaposi sarcoma [86], keratosis lichenoides chronica [87,88], lichen amyloidosis [89], lichen striatus [90], linear atrophoderma of Moulin [91], morphea or linear scleroderma [92][93][94], nevoid hyperkeratosis [95,96], oral leukoplakia [97,98], peeling skin syndrome [99], pityriasis rubra pilaris [100], progressive symmetrical erythrokeratoderma [101], prurigo nodularis [102], scleroderma [103], Sjogren-Larsson syndrome …”

Section: Vitiligomentioning

confidence: 99%