“…In other words, instead of inducing miniaturization with a subsequent prolonged telogen phase, follicles are instead shifted towards terminal anagen hair growth. Acquired localized hypertrichosis has been described in various other settings of dermal injury, including terminal hair growth at the periphery of a burn [7], transient limb hypertrichosis associated with casting [8,9] peri-incisional hypertrichosis following knee surgery or fracture sites [10,11], distal hair growth following lymphadenectomy [12], terminal hair growth at the site of bug bites [13] and verruca vulgaris [14], local reactions to measles [15], smallpox [16] and other vaccines [17], and chronic rubbing, scratching, and biting associated with various clinical situations [18,19]. Common to these widely varied clinical presentations is the hypothesis that local hyperemia or inflammation may lead to localized terminal hair growth.…”