Acute esophageal necrosis (black esophagus) in a 40-year-old man

Abu‐Zaid, Ahmed; Solimanie, Shrouq; Abudan, Zainab; Al–Hussaini, Hussa; Azzam, Ayman; Amin, Tarek

doi:10.5144/0256-4947.2015.80

Cited by 7 publications

(10 citation statements)

References 4 publications

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“…This characteristic endoscopic appearance is sufficient to establish the diagnosis. Additional endoscopic findings may include signs of bleeding (active oozing from distal esophagus [106], blood clots [26] and/or “coffee ground” material in the stomach [85], multiple esophageal ulcerations [106], gastric ulcers [37], duodenal pathology (ulcers, erosions or edema) [39], or gastric outlet obstruction due to gastric volvulus [19].…”

Section: Etiologymentioning

confidence: 99%

Diagnosis and management of acute esophageal necrosis

Dias

Santos-Antunes

Macedo

2019

aog

113

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Acute esophageal necrosis is a rare syndrome classically characterized by a striking endoscopic image of diffuse and circumferential black mucosal discoloration of distal esophagus, with an abrupt transition at the gastroesophageal junction and variable proximal extension. The typical patient is an older male with general debilitation and multiple comorbidities presenting with hematemesis or melena. The pathophysiology usually involves a combination of esophageal ischemia, backflow injury from gastric chemical contents and impaired mucosal reparative mechanisms associated with debilitated physical states. It may arise in the setting of hemodynamic compromise, diabetic ketoacidosis, hypothermia, alcoholic intoxication, trauma, inflammatory diseases, esophageal local infection, solid organ transplantation, postoperative status, drugs or acute gastric outlet obstruction, usually in the background of a chronic debilitating process, where the concurrent presence of multiple risk factors, including diabetes mellitus, hypertension, malnutrition, malignancy or alcohol abuse, places a patient at higher risk. The characteristic endoscopic appearance establishes the diagnosis. Biopsy is supportive but not required. Management is mainly supportive and consists of correcting coexisting conditions, fluid therapy, bowel rest, intravenous proton pump inhibitor therapy and red blood cell transfusion as needed. Although this is a serious life-threatening condition, appropriate treatment may result in a favorable outcome in the majority of patients.

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Section: Etiologymentioning

confidence: 99%

Diagnosis and management of acute esophageal necrosis

Dias

Santos-Antunes

Macedo

2019

aog

113

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“…Of these cases, we managed to extract data from 79 articles having 105 cases of AON (online supplementary table 1). 5–82 Data could not be extracted from (n=13) of the articles containing a total of (n=20) cases 4 83–95. The most common reasons for these were unavailability of complete data or the language being other than English.…”

Section: Discussionmentioning

confidence: 99%

Comprehensive review of acute oesophageal necrosis

Ullah¹,

Mehmood

Micaily

et al. 2019

BMJ Case Rep

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Acute oesophageal necrosis, also known as ‘black oesophagus’, is a rare condition characterised by the black discolouration of the oesophageal mucosa on endoscopy and involves the distal oesophagus in majority of cases but may also extend proximally. A number of conditions are found to be associated with it and it is thought to occur due to a combination of hypovolaemia and inadequate oesophageal protective mucosal barrier function. Gastric secretions may have a direct effect on the oesophageal mucosa. We present a case of a woman who presented with haematemesis and significant hypotension after a session of haemodialysis. Black oesophagus was confirmed on esophagogastroduodenoscopy. She was given two units of packed red blood cells and one unit of platelets, and started on a pantoprazole infusion. However, despite rigorous attempts at resuscitation the patient failed to recover.

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“…Diagnozė grindžiama endoskopinio tyrimo radiniais, o laboratorinių tyrimų rezultatų pokyčiai dažniausiai yra nespecifiniai [1][2][3][4][5][6]. Pradinis gydymas medikamentinis, tačiau, išsivysčius komplikacijoms, pavyzdžiui, stemplės perforacija ar mediastinitas, reikalingi chirurginiai veiksmai [6,11].…”

Section: įVadasunclassified

“…Auksinis standartas diagnozuoti ŪSN yra ezofagogastroduodenoskopija (EFGDS) [15]. Diagnostinis endoskopinio vaizdo požymis -difuziškai, cirkuliariai stemplės gleivinę dengiantis juodas pigmentas, kuris aiškiai baigiasi ties gastroezofagine jungtimi (GEJ) [1][2][3][4][5][6][7][8][9][10][11][12][13][14][15][16][17][18][19]. 92,9 proc.…”

Section: Tyrimo Rezultataiunclassified

“…sergančiųjų -vyrai ir tik 6,2 proc.moterys. Didžiausia ŪSN sergančiųjų dalis yra 60-69 metų [1][2][3][4][5][9][10][11][12][13][14][15][16][17][18][19]. Gautus rezultatus lyginant su kitų literatūros šaltinių duomenimis, reikšmingo skirtumo nepastebėta -vidutinis ligos pasireiškimo amžius siekė 62-67 m., vyriausias pacientas buvo 91 m., 80 proc.…”

Section: Tyrimo Rezultataiunclassified

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Ūminė Stemplės Nekrozė: Literatūros Ir Klinikinių Atvejų Apžvalga

Vindžigalskytė,

Baliūnaitė,

Kiudelis

2024

Health Sciences

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Tyrimo tikslas. Atlikti literatūros šaltinių analizę apie ūminės stemplės nekrozės (ŪSN) etiopatogenezę, klinikinius požymius, diagnostikos ir gydymo galimybes. Metodai. Remiantis Google Scholar, PubMed ir ClinicalKey duomenų bazių šaltiniais, išanalizuota 16 publikacijų pasirinkta tema. Išvados. ŪSN yra daugiaetiologė liga, kliniškai pasireiškianti kraujavimo iš viršutinio virškinamojo trakto požymiais, skausmu epigastrio srityje, karščiavimu, silpnumu. Pagrindinis diagnostikos metodas yra endoskopinis tyrimas. Gydymas apima intraveninę hidrataciją, antacidinius vaistus, parenterinę mitybą, nil per os principą ir komplikacijų valdymą.

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Acute esophageal necrosis (black esophagus) in a 40-year-old man

Cited by 7 publications

References 4 publications

Diagnosis and management of acute esophageal necrosis

Diagnosis and management of acute esophageal necrosis

Comprehensive review of acute oesophageal necrosis

Ūminė Stemplės Nekrozė: Literatūros Ir Klinikinių Atvejų Apžvalga

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