Acute Myocardial Infarction and Cerebral Infarction at Kusatsu-spa.

Kubota, Keiichi; Tamura, Kousei; Take, Hitoshi; Kurabayashi, Hitoshi; Shirakura, Takuo

doi:10.3143/geriatrics.34.23

Cited by 9 publications

(8 citation statements)

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“…18 However, whole-body TP is compromised by the initial tissue injury and cardiovascular changes, limiting its clinical applicability. [19][20][21] Water-bath immersion at 41°C for 15 minutes increases heart rate, 20 and elevation of the core temperature to 42°C for 15 minutes increases polymorphonuclear cell migration to the myocardium. 21 In some humans, hot bathing TP induces acute myocardial and cerebral infarction Յ3 hours, possibly via transient changes in blood pressure, heart rate, blood viscosity, fibrinolytic activity, and platelet function.…”

mentioning

confidence: 99%

“…21 In some humans, hot bathing TP induces acute myocardial and cerebral infarction Յ3 hours, possibly via transient changes in blood pressure, heart rate, blood viscosity, fibrinolytic activity, and platelet function. 19 For prevention of exacerbated hemodynamic variation during TP, a safe strategy for waterbath immersion at 41°C is recommended to be Յ5 minutes and that at 38°C is Յ10 minutes. 20 However, the vascular protection may be attenuated because of short duration and low temperature (Ͻ42°C).…”

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confidence: 99%

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Repetitive progressive thermal preconditioning hinders thrombosis by reinforcing phosphatidylinositol 3-kinase/Akt-dependent heat-shock protein/endothelial nitric oxide synthase signaling

Yang

Hsu

et al. 2012

Journal of Vascular Surgery

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confidence: 99%

mentioning

confidence: 99%

Repetitive progressive thermal preconditioning hinders thrombosis by reinforcing phosphatidylinositol 3-kinase/Akt-dependent heat-shock protein/endothelial nitric oxide synthase signaling

Yang

Hsu

et al. 2012

Journal of Vascular Surgery

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“…1,2 Blood viscosity increases during hot and dry weather, 5 and bathing in hotsprings may cause a transient change of blood viscosity that induces acute cerebral infarction or myocardial infarction. 6 It has also been reported that the infarct rate is higher in the early morning because blood viscosity increases at this time. 7 Based on such findings, many physicians and neurologist (as well as the mass media) have recommended a high water intake as a useful precaution.…”

Section: Discussionmentioning

confidence: 94%

“…It has been reported that blood viscosity is increased in patients with ischemic heart disease or cerebrovascular disease, and that dehydration may be one of the causes of myocardial ischemia or stroke 1,2 . Blood viscosity increases during hot and dry weather, 5 and bathing in hot‐springs may cause a transient change of blood viscosity that induces acute cerebral infarction or myocardial infarction 6 . It has also been reported that the infarct rate is higher in the early morning because blood viscosity increases at this time 7 .…”

Section: Discussionmentioning

confidence: 99%

Change of blood viscosity and urinary frequency by high water intake

et al. 2007

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Physicians recommend a high water intake to prevent cerebral infarction by decreasing blood viscosity. However, there is no evidence that high water intake decreases viscosity, although it increases urinary frequency. Therefore, we investigated the change of blood viscosity and urinary frequency by high water intake. We examined the change of blood viscosity after acute water intake, the circadian rhythm of blood viscosity, and the change of early morning blood viscosity and urinary frequency after high water intake for 1 week in healthy volunteers aged 31-75 years. Blood viscosity decreased transiently by acute water intake, but remained within the range of its circadian rhythm. Morning blood viscosity did not alter by high water intake for 1 week, although urinary frequency increased. Because excessive water intake increases urinary frequency without decreasing blood viscosity, a high water intake should not be recommended in persons with urinary frequency.

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“…Although well tolerated in most cases, there are reports indicating an increased risk of thrombotic events after hot tub bathing (Deguchi et al 1993;Eshel et al 1998;Tamura et al 1996;Kubota et al 1997). Alterations in the haemostatic system have been consistently described in patients presenting with hyperthermia related illnesses such as heat stroke (Bouchama et al 1996;Bouchama and Knochel 2002), or after physical activity such as marathon running, which is also regularly accompanied by increased body temperature (Roecker et al 1990).…”

Section: Introductionmentioning

confidence: 96%

Changes in the haemostatic system after thermoneutral and hyperthermic water immersion

Boldt

Fraszl

Röcker³

et al. 2007

Eur J Appl Physiol

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Warm water bathing is a popular recreational activity and is frequently used in rehabilitation medicine. Although well tolerated in most cases, there are reports indicating an increased risk of thrombotic events after hot tub bathing. The effects of a 45 min thermoneutral bath followed by a 50 min bath with increasing water temperature (maximum 41 degrees C) until reaching a body core temperature of 39 degrees C on factors of blood coagulation and fibrinolysis were studied in eight healthy male volunteers. Blood was obtained after a 45-min resting period as control and after the thermoneutral and hyperthermic bath as well as after another 45 min recovery period at the end of the study. Hyperthermic immersion (HI) lead to a shortening of activated partial thromboplastin time (aPTT) (P < 0.05). Fibrinogen concentration decreased immediately after HI (P < 0.05) but increased during recovery (P < 0.05). Plasminogen activator inhibitor (PAI) activity decreased during HI (P < 0.05), D-dimer concentration was not found to change. Thrombocyte count increased (P < 0.05) during HI. The increases in tissue-type plasminogen activator concentration as well as leucocyte count during HI were due to haemoconcentration. Prothrombin time, PAI-activity and granulocyte count decreased during thermoneutral immersion (P< 0.05). Warm water bathing leads to haemoconcentration and minimal activation of coagulation. The PAI-1 activity is decreased. A marked risk for thrombotic or bleeding complications during warm water bathing in healthy males could not be ascertained.

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Acute Myocardial Infarction and Cerebral Infarction at Kusatsu-spa.

Cited by 9 publications

References 0 publications

Repetitive progressive thermal preconditioning hinders thrombosis by reinforcing phosphatidylinositol 3-kinase/Akt-dependent heat-shock protein/endothelial nitric oxide synthase signaling

Repetitive progressive thermal preconditioning hinders thrombosis by reinforcing phosphatidylinositol 3-kinase/Akt-dependent heat-shock protein/endothelial nitric oxide synthase signaling

Change of blood viscosity and urinary frequency by high water intake

Changes in the haemostatic system after thermoneutral and hyperthermic water immersion

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