Acute Pancreatitis as a Complication of Colonoscopy

Thomas, Andrew W.; Mitre, Ricardo

doi:10.1097/00004836-199409000-00024

Cited by 28 publications

(24 citation statements)

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“…This is the first case of recurrent pancreatitis as a result of colonoscopy that we have been able to find. Two earlier reports describe trauma from passage of the endoscope as probable factors in its development 6 7. Specifically, both these cases describe procedural difficulties, particularly around the splenic flexure.…”

Section: Discussionmentioning

confidence: 99%

Recurrent pancreatitis after unremarkable colonoscopy, temporalised by CT imaging: an unusual case

et al. 2016

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SUMMARYAcute pancreatitis is a common surgical presentation, frequently caused by gallstones and alcohol. Here we present an unusual case of a recurrent episode of pancreatitis after an unremarkable colonoscopy, in a patient with several pre-existing risk factors for pancreatitis. Before and after abdominal CT scans clearly demonstrate the acute inflammatory process affecting the pancreas and temporalise its development. Early resuscitation and appropriate involvement of high dependency care is advocated by all current guidelines to improve patient outcome. We consider possible aetiology and how early diagnosis and recognition of possible high-risk situations can expedite its investigation and management, helping to provide the best possible care. BACKGROUND

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Section: Discussionmentioning

confidence: 99%

Recurrent pancreatitis after unremarkable colonoscopy, temporalised by CT imaging: an unusual case

et al. 2016

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“…The last published case of acute pancreatitis after colonoscopy was that of a 25-year-old man who developed acute pancreatitis 4 hours after undergoing colonoscopy for weight loss and diarrhoea 27. There was some difficulty advancing the scope beyond the splenic flexure, but this was eventually overcome after the scope was withdrawn and reintroduced, allowing visualisation up to the caecum; a 15 mm polyp was removed from the sigmoid colon.…”

Section: Discussionmentioning

confidence: 99%

Acute pancreatitis following oesophagogastroduodenoscopy.

Nwafo¹

2017

BMJ Case Reports

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Acute pancreatitis is a well-recognised complication of endoscopic procedures like endoscopic retrograde cholangiopancreatography but not oesophagogastroduodenoscopy (OGD). I report a case of a 33-year-old woman, admitted with severe epigastric pain and vomiting 2 hours after an elective OGD for evaluation of chronic gastrointestinal symptoms. Pancreatitis was diagnosed on the basis of elevated lipase (40 790 U/L; normal 11-82) and findings on imaging studies. Other common causes of acute pancreatitis such as gallstones, alcohol and medications were ruled out. She had an extended hospital course because of severe disease, characterised by systemic inflammatory response syndrome, pleural effusion and ascites but was successfully managed conservatively with bowel rest, hydration and pain management. Acute pancreatitis should be considered a rare complication of OGD and should be considered in differential diagnosis for abdominal pain post OGD. Pathogenesis is likely from direct trauma to pancreas or gas insufflation.

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“…However, acute pancreatitis as such a complication of push-enteroscopy, caused by an overtube, was described previously by other authors (52). Acute pancreatitis was even described after uneventful upper and lower gastrointestinal endoscopy (53)(54)(55). Blackwood et al detected asymptomatic hyperamylasuria in 6.6% of patients undergoing gastrointestinal endoscopy (56).…”

Section: Figmentioning

confidence: 91%

Risk Factors of Acute Pancreatitis in Oral Double Balloon Enteroscopy

Kopáčová

Bureš

Rejchrt

et al. 2016

Acta Med. (Hradec Kralove, Czech Repub.)

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Summary: Double balloon enteroscopy (DBE) was introduced 15 years ago. The complications of diagnostic DBE are rare, acute pancreatitis is most redoubtable one (incidence about 0.3%). Hyperamylasemia after DBE seems to be a rather common condition respectively. The most probable cause seems to be a mechanical straining of the pancreas. We tried to identify patients in a higher risk of acute pancreatitis after DBE. We investigated several laboratory markers before and after DBE (serum cathepsin B, lactoferrin, E-selectin, SPINK 1, procalcitonin, S100 proteins, alfa-1-antitrypsin, hs-CRP, malondialdehyde, serum and urine amylase and serum lipase). Serum amylase and lipase rose significantly with the maximum 4 hours after DBE. Serum cathepsin and procalcitonin decreased significantly 4 hours after DBE compared to healthy controls and patients values before DBE. Either serum amylase or lipase 4 hours after DBE did not correlate with any markers before DBE. There was a trend for an association between the number of push-and-pull cycles and procalcitonin and urine amylase 4 hours after DBE; between procalcitonin and alfa-1-antitrypsin, cathepsin and hs-CRP; and between E-selectin and malondialdehyde 4 hours after DBE. We found no laboratory markers determinative in advance those patients in a higher risk of acute pancreatitis after DBE.

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Acute Pancreatitis as a Complication of Colonoscopy

Cited by 28 publications

References 0 publications

Recurrent pancreatitis after unremarkable colonoscopy, temporalised by CT imaging: an unusual case

Recurrent pancreatitis after unremarkable colonoscopy, temporalised by CT imaging: an unusual case

Acute pancreatitis following oesophagogastroduodenoscopy.

Risk Factors of Acute Pancreatitis in Oral Double Balloon Enteroscopy

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