Acute Postoperative Glaucoma After Nonocular Surgery Remains a Diagnostic Challenge

Lentschener, Claude; Ghimouz, Abdelmalek; Bonnichon, P; Parc, C.; Ozier, Yves

doi:10.1097/00000539-200204000-00049

Cited by 25 publications

(19 citation statements)

References 9 publications

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“…Moreover, the perioperative period carries the risk of psychological stress and darkness-induced mydriasis may increase the risk of glaucoma attacks. 3,118,122 Owing to the patient's systemic early postoperative condition, the classic signs of ACAG may not be detected and the diagnosis can be delayed.…”

Section: Anesthetic Agentsmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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Numerous systemically used drugs are involved in drug-induced glaucoma. Most reported cases of non-steroidal drug-induced glaucoma are closed-angle glaucoma (CAG). Indeed, many routinely used drugs that have sympathomimetic or parasympatholytic properties can cause pupillary block CAG in individuals with narrow iridocorneal angle. The resulting acute glaucoma occurs much more commonly unilaterally and only rarely bilaterally. CAG secondary to sulfa drugs is a bilateral non-pupillary block type and is due to forward movement of iris-lens diaphragm, which occurs in individuals with narrow or open iridocorneal angle. A few agents, including antineoplastics, may induce open-angle glaucoma. In conclusion, the majority of cases with glaucoma secondary to non-steroidal medications are of the pupillary block closed-angle type and preventable if the at-risk patients are recognized and treated prophylactically.

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Section: Anesthetic Agentsmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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“…It has a weak anticholinergic effect, which can also induce pupillary dilation and acute angle closure glaucoma (2,8,(11)(12)(13) . However, so far there is no sufficient evidence that this class of drug would increase IOP (14) .…”

Section: Discussionmentioning

confidence: 99%

“…Angle-closure glaucoma has been previously reported in patients undergoing general anesthesia for non-ophthalmologic surgery (8,12,(17)(18)(19) . Many factors may contribute to the onset of an acute angle closure attack during anesthesia in predisposed individuals: CO 2 retention (20,21) ; endotracheal intubation associated with laryngospasm and cough leading to IOP augmentation, and the use of anticholinergic and adrenergic drugs.…”

Section: Discussionmentioning

confidence: 99%

Acute angle closure glaucoma following ileostomy surgery

Lopes¹,

Tavares²,

Almeida³

et al. 2015

Revista Brasileira De Oftalmologia

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RESUMOO glaucoma de fechamento angular pode ser induzido por drogas que podem causar dilatação da pupila. Nós relatamos um caso no qual a paciente desenvolveu fechamento angular em ambos os olhos após injeção sistêmica de atropina. Este caso mostra a importância de uma pronta intervenção em doenças com envolvimento ocular com o objetivo de realizar o tratamento adequado. Descritores: Ileostomia/efeitos adversos; Glaucoma de ângulo fechado/etiologia; Glaucoma de ângulo fechado/diagnóstico; Relatos de casos

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“…[1][2][3][4][5] However, the relationship between general anesthesia and postoperative AACG remains unclear.…”

Section: Discussionmentioning

confidence: 99%

“…Sympathomimetics such as phenylephrine can reportedly cause AACG. 5 Some postoperative AACG cases are reportedly related to ephedrine. 3,5 Despite its short vasoactive effect, ephedrine can result in dilatation of the pupil for 3-6 hours.…”

Section: Discussionmentioning

confidence: 99%

Acute Angle-Closure Glaucoma After General Anesthesia for Bone Grafting

Nitta

Kamekura

Takuma

et al. 2014

Anesthesia Progress

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Acute angle-closure glaucoma (AACG) is a rare complication of general anesthesia. The coexistence of individual risk factors for postoperative AACG and factors associated with intraocular hypertension are considered to be required for postoperative AACG to develop. We present a case of AACG after general anesthesia for oral bone grafting in a patient with no preoperative eye symptoms. In this case, several factors such as postoperative care in a darkened room, psychological stress, and postoperative hypertension may have precipitated the event in this patient, who may have had preexisting undiagnosed elevated intraocular pressure. The interval between the earliest appearance of symptoms at 9 hours and the ultimate diagnosis was 36 hours. In the postoperative period following general anesthesia, any patient is at risk for AACG. It is important that a postoperative diagnosis of AACG should be considered and a timely consultation with an ophthalmologist be considered if a postoperative patient complains of red eyes, visual disorder, eye pain, headache, and nausea.

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Acute Postoperative Glaucoma After Nonocular Surgery Remains a Diagnostic Challenge

Cited by 25 publications

References 9 publications

Non-steroidal drug-induced glaucoma

Non-steroidal drug-induced glaucoma

Acute angle closure glaucoma following ileostomy surgery

Acute Angle-Closure Glaucoma After General Anesthesia for Bone Grafting

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