Acute Pulmonary Edema in Battle Casualties

Rl, Simmons; Ca, Heisterkamp; Ja, Collins; Ce, Bredenburg; Am, Martin

doi:10.1097/00005373-196909000-00002

Cited by 30 publications

(6 citation statements)

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“…This experimental pulmonary insufficiency was similar to that ob served earlier in the dog [1] and to human posltraumatic pulmonary insuf ficiency [7,13,18,25]. In the rat, however, the time course is much com pressed and the changes in respiratory function, which in humans occur over a period of 1 or several days and in the dog in 6 h, take place in 60-90 min after induction of intravascular coagulation.…”

Section: Discussionsupporting

confidence: 84%

Pulmonary Insufficiency in the Rat after Intravascular Coagulation and Inhibition of Fibrinolysis

Gerdin¹,

Diffang²,

Saldeen³

1981

Eur Surg Res

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Thrombin-induced intravascular coagulation was followed by pulmonary dysfunction in rats treated with the fibrinolysis inhibitor AMCA (trans-4-aminoethyl-cyclohexane carboxylic acid). The pulmonary dysfunction developed after a latency period of about 60 min and led progressively to pulmonary insufficiency with a morphological and functional pattern similar to that in the human delayed microembolism syndrome. Neither activation of the kallikrein-kinin system nor inhibition of converting enzyme activity in the lungs was of major importance in the pathogenesis. Accumulation of fibrin degradation products (FDP) occurred in the lungs and may have played a casual role.

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Section: Discussionsupporting

confidence: 84%

Pulmonary Insufficiency in the Rat after Intravascular Coagulation and Inhibition of Fibrinolysis

Gerdin¹,

Diffang²,

Saldeen³

1981

Eur Surg Res

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“…Las primeras publicaciones de casos datan de principios del siglo veinte 5 , la mayoría asociados a lesiones cráneo-encefálicas en campo de batalla ocurridos en la Primera Guerra Mundial 6,7 . Actualmente se sabe que el EPN puede ser desencadenado prácticamente por cualquier lesión del sistema nervioso central 2 (Tabla 1).…”

Section: Discussionunclassified

Edema pulmonar neurogénico secundario a crisis epiléptica: Presentación de un caso y revisión de la literatura

A¹,

C²,

A³

et al. 2009

Rev. chil. enferm. respir.

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“…The low p O 2 occurring in wounded patients was confirmed by our shock‐trauma unit in Vietnam, headed in turn by Drs. John A. Collins, Paul M. James, Terruo Matsumoto, Carl Bredenberg, and finally, from 1965 to1968, by Dr. R. L. Simmons 6–14. A low p O 2 in wounded patients was also confirmed by the Navy Hospital at Da Nang in Vietnam, where the condition was called “wet lung” or “Da Nang lung.”11…”

Section: Case No Diagnosis Po2 Outcomementioning

confidence: 93%