The issue of fertility following acute gonococcal salpingitis has been addressed in less than an optimal manner. The often demographic instability of study patients and the need to publish within specific time These studies, done in the 1950s and 1960s, correlate well with our current understanding of the pathogenesis of acute gonococcal salpingitis. 6'7 In the absence of a concomitant Chlamydia trachomatis infection, gonococcal salpingitis is initially monomicrobial in etiology. 6'8-11 With an alteration of the oxidation-reduction potential, the "anaerobic progression" is initiated.9-11 The current theory is that anaerobic bacteria are primarily responsible for basement-membrane destruction and healing by fibrosis within the fallopian tube.9 Early monoetiological gonococcal salpingitis is associated with a relatively limited elevation of