“…Resistant cultivars carry genes with resistance to stalk rot infection, which are transferred via a quantitative inheritance mode, with the predominance of an additive genetic effect (CARSON & HOOKER, 1981;BADU-APRAKU et al, 1987;TOMAN & WHITE, 1993;BERGSTROM & NICHOLSON, 1999;PALAVERSIC et al, 2009;MATIELLO et al, 2012). Maize genotypes showing different levels of anthracnose stalk rot resistance have been described in germplasm banks; examples of such genotypes are the following: MP305, DE811ASR (JUNG et al, 1994, BROGLIE et al, 2006FREY et al, 2011);DW1035 (TOMAM & WHITE, 1993; A556, A638, Oh43, R177 (CARSON & HOOKER, 1981); RD6502 (BADU-APRAKU et al, 1987);Bc19064 (PALAVERSIC et al, 2009);CML52 (CHUNG et al, 2011);Das2, Das64 (MATIELLO et al, 2012); H8664 (MATIELLO et al, 2013); 2B710 (GARDINGO, 2008;COSTA et al, 2010b;COTA et al, 2010;CARVALHO et al, 2013). However, in practice, the resistance levels of these genotypes remain weak, and information on effective resistance sources is lacking.…”