Allergic Vasculitis Gougerot-Ruiter Syndrome

Stringa, S. G.; Bianchi, C; Casalá, A. M.; Bianchi, O

doi:10.1001/archderm.1967.01600310029005

Cited by 28 publications

(3 citation statements)

References 16 publications

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“…The granular deposits of immunoglobulins, as sociated with C3, in the blood vessel walls are high ly suggestive evidence of tissue-fixed immune com plexes. Although some authors (6,9,10) reported a predominance of IgG as the deposited immuno globulin, a recent report by Sams et al (8) stated that lgM and lgA were two major immunoglobulins. Our present study has confirmed this.…”

Section: Healthy Controlsmentioning

confidence: 97%

Demonstration of circulating and tissue-fixed immune complexes in cutaneous necrotizing vasculitis

Imamura¹,

Yanase²

1980

Acta Derm Venereol

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Simultaneous demonstration of circulating and tissue-fixed immune complexes was attempted in 22 patients with cutaneous necrotizing vasculitis (7 anaphylactoid purpura, 9 cutaneous allergic vasculitis, 2 livedo reticularis, 1 thrombophlebitis, 2 erythema elevatum diutinum and 1 acute generalized pustular bacterid). In 16 out of the 22 patients, particularly patients with anaphylactoid purpura and cutaneous necrotizing vasculitis, there was a high Clq-binding activity. Decreased levels of C3 and C4 were seen in 2 and 3 patients, respectively. In 11 out of 16 skin lesions, the granular deposits of immunoglobulins and/or complement were demonstrated in the blood vessel walls of the dermis. IgA deposit was seen in anaphylactoid purpura, and IgM deposit in other types of vasculitis. C3 deposit was the most frequently noted. There was no definite correlation between Clq-binding activity and tissue deposits.

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Section: Healthy Controlsmentioning

confidence: 97%

Demonstration of circulating and tissue-fixed immune complexes in cutaneous necrotizing vasculitis

Imamura¹,

Yanase²

1980

Acta Derm Venereol

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“…Die Hautbefunde bei kutanen Vaskulitiden sind in Übersichtsartikeln [3,4,10,11,18,31,35,36] und in dermatologischen Lehrbüchern [5,28,33] bestens und ausführlich beschrieben. Die .…”

Section: Hautmanifestationen Bei Kutanen Vaskulitidenunclassified

“…Nur die leukozytoklastische Vaskulitis und ihre Unterformen zeigen obligat Hautveränderungen Von allen in der CHC berücksichtigten Erkrankungen zeigen nur die leukozytoklastische Vaskulitis (LcV) und ihre Unterformen obligat Hautveränderungen. Die Krankheitsbezeichnung "leukozytoklastische Vaskulitis" wird in der deutschsprachigen Dermatologie synonym mit "Vasculitis allergica, Immunkomplexvaskulitis oder auch nekrotisierende Vaskulitis" verwendet, obwohl weder Leukozytoklasie noch Allergien, Immunkomplexe oder Hautnekrosen bei allen Patienten mit LcV nachweisbar sein müssen [33,34]. Der im englischsprachigen Raum eingeführte Begriff "cutaneous small vessel vasculitis" (CSVV) ist zutreffender, aber in der deutschen Übersetzung nicht gebräuchlich [4,5,28].…”

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Kutane Vaskulitiden

Schäkel

Meurer

2008

Hautarzt

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Vasculitis is characterized by an inflammatory reaction of vessel walls with damage to the dependent tissues. Forms of vasculitis which frequently have skin changes include leukocytoclastic angiitis (LcV), Henoch-Schönlein purpura (HSP), cutaneous polyarteriitis nodosum (cPAN), erythema elevatum et diutinum (EED) and urticarial vasculitis (UV). In other forms of vasculitis, systemic manifestations predominate but there are a variety of skin changes. Kawasaki disease (MK), cryoglobulinemic vasculitis (kV), Wegener granulomatosis (WG), Churg-Strauss syndrome (CSS) and microscopic polyangitis (MPA) belong to this group. The causes of vasculitis are heterogeneous. Triggers include infections, drugs, collagen vascular diseases, autoimmune diseases and lymphoproliferative disorders. Idiopathic vasculitis, particularly LcV and EED, occur only once and have a self-limited course. The diagnostic work up depends on the clinical picture and includes inflammatory markers, circulating immune complexes, different types of cryoglobulins and anti-neutrophilic cytoplasmic antibodies, collagen vascular disease specific autoantibodies and additional hematological studies. Vasculitis can manifest in many organs and requires a thorough work up specifically in cases where WG, MPA, CSS and PAN are under consideration.

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