Alterations of Fibrinolysis and Blood Coagulation

Mannucci, P. M.; Lobina, G. F.; Ruggeri, Zaverio M.

doi:10.1016/s0140-6736(69)91505-0

Cited by 3 publications

(1 citation statement)

References 3 publications

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“…Though disturbances of function have been shown to occur in several organs and systems after pneumoencephalography (Boudin et al, 1954;Jorgensen, 1957;Wender, 1957;Vara L6pez and Duran Sacristan, 1958;Davie, 1963;Hudson et al, 1967;Puzyniski, 1967;Mannucci et al, 1969) no significant alteration of plasma potassium concentration had previously been reported. Wise et al (1958) could not find any significant change in their three patients, but the measurement of serum electrolytes, though not clearly stated, seems to have been recorded 24 hours after pneumoencepholography had been carried out.…”

Section: Discussionmentioning

confidence: 99%

Hypokalaemia after Pneumoencephalography

Podda¹,

Agostoni²,

Pintus³

et al. 1971

BMJ

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SummaryThe changes in plasma electrolytes and blood pH and gases following the insufflation of air into the subarachnoid space for pneumoencephalography were investigated. The only significant change observed was a rapid fall of plasma potassium most evident at 60 minutes. A concomitant reduction of the urinary Na/K ratio supports the possibility that the change observed may be due to the stimulation of aldosterone secretion caused by this procedure, though its rapid occurrence and reversibility indicate that other influences might at least in part be responsible.

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Section: Discussionmentioning

confidence: 99%

Hypokalaemia after Pneumoencephalography

Podda¹,

Agostoni²,

Pintus³

et al. 1971

BMJ

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Die Pneumographie

Thun¹

1977

Handbuch Der Medizinischen Radiologie / Encyclopedia of Medical Radiology

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Adrenergic Mechanisms in the Systemic Plasminogen Activator Response to Adrenaline in Man

1970

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Summary. The effect of propranolol, ICI 50172 and a combination of propranolol and phentolamine, on the plasminogen activator response to a 30 mm intravenous infusion of adrenaline hydrochloride (0.1 μg/kg/min) has been studied in five healthy subjects. Plasma factor VIII, serum free fatty acids, blood glucose and pulse rate changes were also monitored to confirm the efficacy of adrenergic blockade. Propranolol blocked the plasminogen activator response by a factor of 29% whereas ICI 50172 had no effect. The addition of phentolamine to propranolol failed to influence further the degree of blockade already present with propranolol alone. It is postulated that the plasminogen activator response to adrenaline is derived from two separate components, and that the relatively minor one may be secondary to vasoactive changes whereas the second and major component is quite independent of this phenomenon. It is also proposed that both components may share a common adrenergic receptor site.

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Alterations of Fibrinolysis and Blood Coagulation

Cited by 3 publications

References 3 publications

Hypokalaemia after Pneumoencephalography

Hypokalaemia after Pneumoencephalography

Die Pneumographie

Adrenergic Mechanisms in the Systemic Plasminogen Activator Response to Adrenaline in Man

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