Background: Arthroscopic Bankart repair is currently the most utilized technique in the surgical management of symptomatic anterior shoulder instability. Despite a growing body of literature regarding optimal repair configurations, little is known about inferior suture anchor placement (6 o'clock position). Methods: 12 cadaveric shoulders were tested on a 6 DOF robotic musculoskeletal simulator to measure the peak resistance force due to anterior displacement of 1 cm. The rotator cuff muscles were loaded during testing to simulate physiological conditions. Test conditions consisted of the intact shoulder, Bankart lesion, Bankart repair (3, 4, and 5 o'clock anchors), and Bankart repair with addition of a 6 o'clock anchor. A 13% anterior bone defect was then and all conditions were repeated. Repeated measures ANOVA was used to test for significant differences among groups. Results: In the no bone loss group, the addition of a 6 o'clock anchor yielded the highest peak resistance force (52.8N, SD: 4.5N) and was significantly greater than the standard Bankart repair by 15.8% (7.2N, p = 0.003). With 13% bone loss from the anterior glenoid, both the standard Bankart repair (peak force 49.3N, SD: 6.1N, p = 0.02) and repair with the addition of the 6 o'clock anchor (peak force 52.6N, SD: 6.1N, p = 0.006) had a significantly higher peak resistance force compared to the bone loss with Bankart lesion group (35.2N, SD: 5.8N). While the 6 o'clock anchor did increase the strength of the standard repair by 6.7%, this was not statistically significant (p = 0.9) in the bone loss model. Conclusions: The addition of a 6 o'clock suture anchor to a "standard" Bankart repair increases the peak resistance force to displacement (no bone loss), although this additional strength is lost with creation of a subcritical anterior glenoid bone defect. Clinical Relevance: This study provides surgeons with essential biomechanical data to aid in the selection of repair configuration. The six o'clock anchor does not enhance repair strength in a subcritical bone loss model.